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Advances in Experimental Medicine and Biology|November 26, 2013
Hypoxic VDAC1: a potential mitochondrial marker for cancer therapyM Christiane Brahimi-Horn, N M MazureBulletin Du Cancer|May 26, 2011
Hypoxic mitochondria: accomplices in resistanceN M Mazure, M C Brahimi-Horn, J PouysségurTumour Biology : the Journal of the International Society for Oncodevelopmental Biology and Medicine|September 13, 2001
Severe hypoxia specifically downregulates hepatocyte nuclear factor-4 gene expression in HepG2 human hepatoma cellsN M Mazure, T L Nguyen, J L DananCurrent Pharmaceutical Design|February 7, 2003
Protein kinases and the hypoxia-inducible factor-1, two switches in angiogenesisN M Mazure, M C Brahimi-Horn, J PouysségurBlood|November 14, 1997
Induction of vascular endothelial growth factor by hypoxia is modulated by a phosphatidylinositol 3-kinase/Akt signaling pathway in Ha-ras-transformed cells through a hypoxia inducible factor-1 transcriptional elementN M Mazure, E Y Chen, K R Laderoute, et al.Cancer Research|April 6, 2001
Hypoxia activates a platelet-derived growth factor receptor/phosphatidylinositol 3-kinase/Akt pathway that results in glycogen synthase kinase-3 inactivationE Y Chen, N M Mazure, J A Cooper, et al.Cancer Research|August 1, 1996
Oncogenic transformation and hypoxia synergistically act to modulate vascular endothelial growth factor expressionN M Mazure, E Y Chen, P Yeh, et al.Oncogene|October 18, 2011
The asparaginyl hydroxylase factor-inhibiting HIF is essential for tumor growth through suppression of the p53-p21 axisJ Pelletier, F Dayan, J Durivault, et al.Pageof 2