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N Miyamura

Showing results (21-30 of 26) with videos related to

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Diabetologia|April 1, 1995
Expression of insulin receptor on clonal pancreatic alpha cells and its possible role for insulin-stimulated negative regulation of glucagon secretionK Kisanuki, H Kishikawa, E Araki, et al.
The Journal of Clinical Investigation|October 15, 1996
A carboxy-terminal truncation of human alpha-galactosidase A in a heterozygous female with Fabry disease and modification of the enzymatic activity by the carboxy-terminal domain. Increased, reduced, or absent enzyme activity depending on number of amino acid residues deletedN Miyamura, E Araki, K Matsuda, et al.
The Journal of Endocrinology|August 15, 2002
Heterozygous knockout of the IRS-1 gene in mice enhances obesity-linked insulin resistance: a possible model for the development of type 2 diabetesA Shirakami, T Toyonaga, K Tsuruzoe, et al.
Diabetologia|April 1, 1996
Bradykinin enhances GLUT4 translocation through the increase of insulin receptor tyrosine kinase in primary adipocytes: evidence that bradykinin stimulates the insulin signalling pathwayS Isami, H Kishikawa, E Araki, et al.
Diabetologia|May 1, 1996
Molecular scanning of the insulin receptor substrate-1 (IRS-1) gene in Japanese patients with NIDDM: identification of five novel polymorphismsS Ura, E Araki, H Kishikawa, et al.
Clinical Nephrology|October 26, 2005
Identification of Fabry's disease by the screening of alpha-galactosidase A activity in male and female hemodialysis patientsM Tanaka, T Ohashi, M Kobayashi, et al.
Pageof 3

Showing results (21-30 of 26) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 26 results.
Diabetologia|April 1, 1995
Expression of insulin receptor on clonal pancreatic alpha cells and its possible role for insulin-stimulated negative regulation of glucagon secretionK Kisanuki, H Kishikawa, E Araki, et al.
The Journal of Clinical Investigation|October 15, 1996
A carboxy-terminal truncation of human alpha-galactosidase A in a heterozygous female with Fabry disease and modification of the enzymatic activity by the carboxy-terminal domain. Increased, reduced, or absent enzyme activity depending on number of amino acid residues deletedN Miyamura, E Araki, K Matsuda, et al.
The Journal of Endocrinology|August 15, 2002
Heterozygous knockout of the IRS-1 gene in mice enhances obesity-linked insulin resistance: a possible model for the development of type 2 diabetesA Shirakami, T Toyonaga, K Tsuruzoe, et al.
Diabetologia|April 1, 1996
Bradykinin enhances GLUT4 translocation through the increase of insulin receptor tyrosine kinase in primary adipocytes: evidence that bradykinin stimulates the insulin signalling pathwayS Isami, H Kishikawa, E Araki, et al.
Diabetologia|May 1, 1996
Molecular scanning of the insulin receptor substrate-1 (IRS-1) gene in Japanese patients with NIDDM: identification of five novel polymorphismsS Ura, E Araki, H Kishikawa, et al.
Clinical Nephrology|October 26, 2005
Identification of Fabry's disease by the screening of alpha-galactosidase A activity in male and female hemodialysis patientsM Tanaka, T Ohashi, M Kobayashi, et al.
Pageof 3