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Proceedings of the National Academy of Sciences of the United States of America|February 1, 1980
Isolation and characterization of polyoma virus mutants able to develop in embryonal carcinoma cellsM Vasseur, C Kress, N Montreau, et al.The EMBO Journal|December 1, 1993
Transcription enhancer factor-1 (TEF-1) DNA binding sites can specifically enhance gene expression at the beginning of mouse developmentF Mélin, M Miranda, N Montreau, et al.Nature|April 23, 1981
Polyoma DNA sequences involved in control of viral gene expression in murine embryonal carcinoma cellsM Katinka, M Vasseur, N Montreau, et al.The EMBO Journal|July 1, 1985
Common features of polyomavirus mutants selected on PCC4 embryonal carcinoma cellsF Melin, H Pinon, C Reiss, et al.Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|April 1, 1999
Rat embryo fibroblasts transformed by c-Jun display highly metastatic and angiogenic activities in vivo and deregulate gene expression of both angiogenic and antiangiogenic factorsM Kraemer, R Tournaire, V Dejong, et al.Molecular and Cellular Biology|May 1, 1996
Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalizationL Vandel, N Montreau, E Vial, et al.Molecular and Cellular Biology|June 1, 1997
The c-Jun-induced transformation process involves complex regulation of tenascin-C expressionA Mettouchi, F Cabon, N Montreau, et al.The EMBO Journal|December 1, 1994
SPARC and thrombospondin genes are repressed by the c-jun oncogene in rat embryo fibroblastsA Mettouchi, F Cabon, N Montreau, et al.Oncogene|November 13, 2001
The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation siteF Bost, L Caron, E Vial, et al.Pageof 1