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N Ninkina

Showing results (31-40 of 56) with videos related to

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Molekuliarnaia Biologiia|August 15, 2012
[Proteinopathies--forms of neurodegenerative disorders with protein aggregation-based pathology]T A Shelkovnikova, A A Kulikova, F O Tsvetkov, et al.
Brain Research. Molecular Brain Research|November 1, 1995
Nerve growth factor-regulated properties of sensory neurones in Oct-2 null mutant miceN N Ninkina, V L Buchman, A N Akopian, et al.
Molecular and Cellular Neurosciences|April 1, 1996
Intracellular compartmentalization of two differentially spliced s-rex/NSP mRNAs in neuronsI D Baka, N N Ninkina, L G Pinon, et al.
Biomedicines|September 23, 2022
Sex-Related Differences in Voluntary Alcohol Intake and mRNA Coding for Synucleins in the Brain of Adult Rats Prenatally Exposed to AlcoholViktor S Kokhan, Kirill Chaprov, Natalia N Ninkina, et al.
Acta Naturae|February 5, 2019
Gamma-Carbolines Derivatives As Promising Agents for the Development of Pathogenic Therapy for ProteinopathyV I Skvortsova, S O Bachurin, A A Ustyugov, et al.
Neuron|September 1, 1991
The octamer-binding protein Oct-2 represses HSV immediate-early genes in cell lines derived from latently infectable sensory neuronsK A Lillycrop, C L Dent, S C Wheatley, et al.
The Journal of Biological Chemistry|September 5, 1992
Regulation of expression of the neuronal POU protein Oct-2 by nerve growth factorJ N Wood, K A Lillycrop, C L Dent, et al.
Neuro-Degenerative Diseases|May 18, 2011
Dimebon does not ameliorate pathological changes caused by expression of truncated (1-120) human alpha-synuclein in dopaminergic neurons of transgenic miceTatyana A Shelkovnikova, Alexey A Ustyugov, Steven Millership, et al.
Biomeditsinskaia Khimiia|July 15, 2014
[Study into molecular targets of a neuroprotective compound dimebon using a transgenic mice line]T A Shelkovnikova, A A Ustiugov, V S Kokhan, et al.
Neuroscience Letters|October 26, 1999
Mutations in the gene encoding human persyn are not associated with amyotrophic lateral sclerosis or familial Parkinson's diseaseJ M Flowers, P N Leigh, A M Davies, et al.
Pageof 6

Showing results (31-40 of 56) with videos related to

Sort By:
Pageof 6
Molekuliarnaia Biologiia|August 15, 2012
[Proteinopathies--forms of neurodegenerative disorders with protein aggregation-based pathology]T A Shelkovnikova, A A Kulikova, F O Tsvetkov, et al.
Brain Research. Molecular Brain Research|November 1, 1995
Nerve growth factor-regulated properties of sensory neurones in Oct-2 null mutant miceN N Ninkina, V L Buchman, A N Akopian, et al.
Molecular and Cellular Neurosciences|April 1, 1996
Intracellular compartmentalization of two differentially spliced s-rex/NSP mRNAs in neuronsI D Baka, N N Ninkina, L G Pinon, et al.
Biomedicines|September 23, 2022
Sex-Related Differences in Voluntary Alcohol Intake and mRNA Coding for Synucleins in the Brain of Adult Rats Prenatally Exposed to AlcoholViktor S Kokhan, Kirill Chaprov, Natalia N Ninkina, et al.
Acta Naturae|February 5, 2019
Gamma-Carbolines Derivatives As Promising Agents for the Development of Pathogenic Therapy for ProteinopathyV I Skvortsova, S O Bachurin, A A Ustyugov, et al.
Neuron|September 1, 1991
The octamer-binding protein Oct-2 represses HSV immediate-early genes in cell lines derived from latently infectable sensory neuronsK A Lillycrop, C L Dent, S C Wheatley, et al.
The Journal of Biological Chemistry|September 5, 1992
Regulation of expression of the neuronal POU protein Oct-2 by nerve growth factorJ N Wood, K A Lillycrop, C L Dent, et al.
Neuro-Degenerative Diseases|May 18, 2011
Dimebon does not ameliorate pathological changes caused by expression of truncated (1-120) human alpha-synuclein in dopaminergic neurons of transgenic miceTatyana A Shelkovnikova, Alexey A Ustyugov, Steven Millership, et al.
Biomeditsinskaia Khimiia|July 15, 2014
[Study into molecular targets of a neuroprotective compound dimebon using a transgenic mice line]T A Shelkovnikova, A A Ustiugov, V S Kokhan, et al.
Neuroscience Letters|October 26, 1999
Mutations in the gene encoding human persyn are not associated with amyotrophic lateral sclerosis or familial Parkinson's diseaseJ M Flowers, P N Leigh, A M Davies, et al.
Pageof 6