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Journal of Endocrinological Investigation|June 27, 2013
Multiple endocrine neoplasia syndromes associated with mutation of p27M Lee, N S PellegataOncogene|July 20, 1995
The basic carboxy-terminal domain of human p53 is dispensable for both transcriptional regulation and inhibition of tumor cell growthN S Pellegata, J F Cajot, E J StanbridgeEndocrinology|November 1, 1995
Coordinated effects of insulin-like growth factor I on inhibitory pathways of cell cycle progression in cultured cardiac muscle cellsW H Chen, N S Pellegata, P H WangProceedings of the National Academy of Sciences of the United States of America|December 24, 1996
DNA damage and p53-mediated cell cycle arrest: a reevaluationN S Pellegata, R J Antoniono, J L Redpath, et al.Histopathology|July 2, 2004
Hashimoto's thyroiditis with papillary thyroid carcinoma (PTC)-like nuclear alterations express molecular markers of PTCM L Prasad, Y Huang, N S Pellegata, et al.Cancer Research|December 15, 1990
Heterogeneous protooncogene amplification correlates with tumor progression and presence of metastases in gastric cancer patientsG N Ranzani, N S Pellegata, C Previderè, et al.Human Genetics|October 1, 1993
Loss of heterozygosity and K-ras gene mutations in gastric cancerG N Ranzani, B Renault, N S Pellegata, et al.Virchows Archiv : an International Journal of Pathology|January 1, 1994
Primary retroperitoneal mucinous cystoadenocarcinomas: an immunohistochemical and molecular studyP Tenti, S Romagnoli, N S Pellegata, et al.Cancer Research|March 15, 1994
K-ras and p53 gene mutations in pancreatic cancer: ductal and nonductal tumors progress through different genetic lesionsN S Pellegata, F Sessa, B Renault, et al.Anticancer Research|September 1, 1992
Detection of K-ras mutations by denaturing gradient gel electrophoresis (DGGE): a study on pancreatic cancerN S Pellegata, M Losekoot, R Fodde, et al.Pageof 3