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Circulation|February 18, 1997
Disturbed connexin43 gap junction distribution correlates with the location of reentrant circuits in the epicardial border zone of healing canine infarcts that cause ventricular tachycardiaN S Peters, J Coromilas, N J Severs, et al.Circulation|February 27, 2001
Mechanisms of resetting reentrant circuits in canine ventricular tachycardiaM S Hanna, J Coromilas, M E Josephson, et al.Circulation|December 13, 1997
Mechanisms causing sustained ventricular tachycardia with multiple QRS morphologies: results of mapping studies in the infarcted canine heartC Costeas, N S Peters, B Waldecker, et al.Circulation Research|February 19, 1998
Characteristics of the temporal and spatial excitable gap in anisotropic reentrant circuits causing sustained ventricular tachycardiaN S Peters, J Coromilas, M S Hanna, et al.Journal of Electrocardiology|January 10, 2002
Remodeling of cardiac gap junctions: the relationship to the genesis of ventricular tachycardiaA L WitCardiology Clinics|August 1, 1990
Cellular electrophysiologic mechanisms of cardiac arrhythmiasA L WitClinical Physiology and Biochemistry|January 1, 1985
Electrophysiological basis for antiarrhythmic drug actionA L WitClinical Science (London, England : 1979)|June 1, 1996
New insights into myocardial arrhythmogenesis: distribution of gap-junctional coupling in normal, ischaemic and hypertrophied human heartsN S PetersMicroscopy Research and Technique|August 1, 1995
Myocardial gap junction organization in ischemia and infarctionN S PetersCardiology Clinics|December 24, 1997
Cellular electrophysiologic mechanisms of cardiac arrhythmiasC Cabo, A L WitPageof 11