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The American Journal of Cardiology|November 26, 1993
Role of alterations in refractoriness and conduction in the genesis of reentrant arrhythmias. Implications for antiarrhythmic effects of class III drugsA L Wit, J CoromilasAmerican Heart Journal|August 1, 1983
Comparison of the direct effects of nifedipine and verapamil on the electrical activity of the sinoatrial and atrioventricular nodes of the rabbit heartW Ning, A L WitCirculation Research|July 1, 1984
The time course of action potential repolarization affects delayed afterdepolarization amplitude in atrial fibers of the canine coronary sinusB Henning, A L WitThe American Journal of Physiology|July 1, 1987
Effects of reducing [Na+]o on catecholamine-induced delayed afterdepolarizations in atrial cellsG N Tseng, A L WitAmerican Heart Journal|October 1, 1983
Pathophysiologic mechanisms of cardiac arrhythmiasA L Wit, M R RosenThe American Journal of Cardiology|April 30, 1987
Arrhythmogenic actions of antiarrhythmic drugsM R Rosen, A L WitCirculation Research|February 1, 1976
Triggered activity in cardiac muscle fibers of the simian mitral valveA L Wit, P F CranefieldThe American Journal of Physiology|July 1, 1978
Reentrant excitation as a cause of cardiac arrhythmiasA L Wit, P F CranefieldJournal of Molecular and Cellular Cardiology|November 1, 1987
Characteristics of a transient inward current that causes delayed afterdepolarizations in atrial cells of the canine coronary sinusG N Tseng, A L WitPageof 11