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Circulation Research|February 1, 1976
Triggered activity in cardiac muscle fibers of the simian mitral valveA L Wit, P F CranefieldThe American Journal of Physiology|July 1, 1978
Reentrant excitation as a cause of cardiac arrhythmiasA L Wit, P F CranefieldCirculation|November 9, 2000
New mechanism of antiarrhythmic drug action: increasing L-type calcium current prevents reentrant ventricular tachycardia in the infarcted canine heartC Cabo, H Schmitt, A L WitThe American Journal of Cardiology|April 1, 1982
Method for evaluating the effects of antiarrhythmic drugs on ventricular tachycardias with different electrophysiologic characteristics and different mechanisms in the infarcted canine heartJ Davis, R Glassman, A L WitInternational Journal of Cardiac Imaging|January 1, 1986
Mapping impulse propagation in the heart: an approach to the study of arrhythmogenesisA L Wit, A A Tischler, S DillonJournal of the American College of Cardiology|February 14, 1998
Mechanisms for spontaneous termination of monomorphic, sustained ventricular tachycardia: results of activation mapping of reentrant circuits in the epicardial border zone of subacute canine infarctsH Schmitt, A L Wit, J Coromilas, et al.Circulation Research|September 1, 1978
Actions of lidocaine on transmembrane potentials of subendocardial Purkinje fibers surviving in infarcted canine heartsJ D Allen, F J Brennan, A L WitCirculation Research|July 1, 1978
The effects of acetylcholine on the electrical activity of canine cardiac Purkinje fibersD C Gadsby, A L Wit, P F CranefieldCirculation Research|October 1, 1981
Electrogenic sodium extrusion can stop triggered activity in the canine coronary sinusA L Wit, P F Cranefield, D C GadsbyPageof 11