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N Sabbagh

Showing results (101-110 of 330) with videos related to

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Human Genetics|May 1, 1996
An additional allelic variant of the CYP2D6 gene causing impaired metabolism of sparteineD Marez, M Legrand, N Sabbagh, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|December 1, 2001
Correlation of nicotinic receptor binding with clinical and neuropathological changes in Alzheimer's disease and dementia with Lewy bodiesM N Sabbagh, R T Reid, L A Hansen, et al.
Progress in Neuro-Psychopharmacology & Biological Psychiatry|July 23, 2019
Oxidative metabolism alterations in the emotional brain of anxiety-prone ratsChelsea R McCoy, Mohammad N Sabbagh, Jonathan P Huaman, et al.
The Journal of Nutrition, Health & Aging|December 11, 2002
HMG-CoA reductase inhibitors (statins) in the treatment of Alzheimer's disease and why it would be ill-advise to use one that crosses the blood-brain barrierD L Sparks, D J Connor, P J Browne, et al.
Dementia and Geriatric Cognitive Disorders|July 30, 2002
Progressive aphasia with Lewy bodiesRichard J Caselli, Thomas G Beach, Lucia I Sue, et al.
Archives of Neurology|December 11, 1999
Neurochemical markers do not correlate with cognitive decline in the Lewy body variant of Alzheimer diseaseM N Sabbagh, J Corey-Bloom, P Tiraboschi, et al.
Current Alzheimer Research|April 14, 2006
Studies on the effect of the apolipoprotein E genotype on the lipid profile in Alzheimer's diseaseMarwan N Sabbagh, Shawn Sandhu, Heather Kolody, et al.
Acta Neuropathologica|February 4, 2010
Response to Parkinnen et al. and JellingerThomas Gerald Beach, Charles L White, Marwan N Sabbagh, et al.
Frontiers in Aging Neuroscience|April 2, 2024
Altered resting-state functional connectivity and dynamic network properties in cognitive impairment: an independent component and dominant-coactivation pattern analyses studyMaurizio Bergamino, Anna Burke, Marwan N Sabbagh, et al.
BMC Neurology|September 28, 2018
Salivary beta amyloid protein levels are detectable and differentiate patients with Alzheimer's disease dementia from normal controls: preliminary findingsMarwan N Sabbagh, Jiong Shi, Moonhee Lee, et al.
Pageof 33

Showing results (101-110 of 330) with videos related to

Sort By:
Pageof 33
Human Genetics|May 1, 1996
An additional allelic variant of the CYP2D6 gene causing impaired metabolism of sparteineD Marez, M Legrand, N Sabbagh, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|December 1, 2001
Correlation of nicotinic receptor binding with clinical and neuropathological changes in Alzheimer's disease and dementia with Lewy bodiesM N Sabbagh, R T Reid, L A Hansen, et al.
Progress in Neuro-Psychopharmacology & Biological Psychiatry|July 23, 2019
Oxidative metabolism alterations in the emotional brain of anxiety-prone ratsChelsea R McCoy, Mohammad N Sabbagh, Jonathan P Huaman, et al.
The Journal of Nutrition, Health & Aging|December 11, 2002
HMG-CoA reductase inhibitors (statins) in the treatment of Alzheimer's disease and why it would be ill-advise to use one that crosses the blood-brain barrierD L Sparks, D J Connor, P J Browne, et al.
Dementia and Geriatric Cognitive Disorders|July 30, 2002
Progressive aphasia with Lewy bodiesRichard J Caselli, Thomas G Beach, Lucia I Sue, et al.
Archives of Neurology|December 11, 1999
Neurochemical markers do not correlate with cognitive decline in the Lewy body variant of Alzheimer diseaseM N Sabbagh, J Corey-Bloom, P Tiraboschi, et al.
Current Alzheimer Research|April 14, 2006
Studies on the effect of the apolipoprotein E genotype on the lipid profile in Alzheimer's diseaseMarwan N Sabbagh, Shawn Sandhu, Heather Kolody, et al.
Acta Neuropathologica|February 4, 2010
Response to Parkinnen et al. and JellingerThomas Gerald Beach, Charles L White, Marwan N Sabbagh, et al.
Frontiers in Aging Neuroscience|April 2, 2024
Altered resting-state functional connectivity and dynamic network properties in cognitive impairment: an independent component and dominant-coactivation pattern analyses studyMaurizio Bergamino, Anna Burke, Marwan N Sabbagh, et al.
BMC Neurology|September 28, 2018
Salivary beta amyloid protein levels are detectable and differentiate patients with Alzheimer's disease dementia from normal controls: preliminary findingsMarwan N Sabbagh, Jiong Shi, Moonhee Lee, et al.
Pageof 33