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Human Molecular Genetics|December 1, 2001
Mice transgenic for the human myotonic dystrophy region with expanded CTG repeats display muscular and brain abnormalitiesH Seznec, O Agbulut, N Sergeant, et al.
Molecular Psychiatry|April 27, 2016
Functional screening of Alzheimer risk loci identifies PTK2B as an in vivo modulator and early marker of Tau pathologyP Dourlen, F J Fernandez-Gomez, C Dupont, et al.
Biochimica Et Biophysica Acta|January 21, 2014
Tau exon 2 responsive elements deregulated in myotonic dystrophy type I are proximal to exon 2 and synergistically regulated by MBNL1 and MBNL2C Carpentier, D Ghanem, F J Fernandez-Gomez, et al.
Molecular Psychiatry|December 3, 2014
A2A adenosine receptor deletion is protective in a mouse model of TauopathyC Laurent, S Burnouf, B Ferry, et al.
Experimental Neurology|January 8, 2008
Overexpression of MBNL1 fetal isoforms and modified splicing of Tau in the DM1 brain: two individual consequences of CUG trinucleotide repeatsC M Dhaenens, S Schraen-Maschke, H Tran, et al.
Biochimica Et Biophysica Acta|March 29, 2011
Mis-splicing of Tau exon 10 in myotonic dystrophy type 1 is reproduced by overexpression of CELF2 but not by MBNL1 silencingC M Dhaenens, H Tran, M-L Frandemiche, et al.
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