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American Journal of Human Genetics|February 15, 2001
Homozygosity mapping of Portuguese and Japanese forms of ataxia-oculomotor apraxia to 9p13, and evidence for genetic heterogeneityM C Moreira, C Barbot, N Tachi, et al.Human Mutation|January 1, 1997
Facilitated diagnosis of CMT1A duplication in chromosome 17p11.2-12: analysis with a CMT1A-REP repeat probe and photostimulated luminescence imagingT Ikegami, H Ikeda, P F Chance, et al.Journal of Medical Virology|August 1, 1993
Epstein-Barr virus genomic sequences and specific antibodies in cerebrospinal fluid in children with neurologic complications of acute and reactivated EBV infectionsS Imai, N Usui, M Sugiura, et al.Nature Genetics|November 1, 1993
De novo mutation of the myelin P0 gene in Dejerine-Sottas disease (hereditary motor and sensory neuropathy type III)K Hayasaka, M Himoro, Y Sawaishi, et al.Human Molecular Genetics|February 28, 1998
Fine mapping of de novo CMT1A and HNPP rearrangements within CMT1A-REPs evidences two distinct sex-dependent mechanisms and candidate sequences involved in recombinationJ Lopes, N Ravisé, A Vandenberghe, et al.Nature Genetics|October 5, 2001
The gene mutated in ataxia-ocular apraxia 1 encodes the new HIT/Zn-finger protein aprataxinM C Moreira, C Barbot, N Tachi, et al.Leukemia|April 8, 2017
Myeloproliferative leukemia protein activation directly induces fibrocyte differentiation to cause myelofibrosisT Maekawa, Y Osawa, T Izumi, et al.Leukemia|September 21, 2018
Correction: Myeloproliferative leukemia protein activation directly induces fibrocyte differentiation to cause myelofibrosisT Maekawa, Y Osawa, T Izumi, et al.Pageof 10