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N Zamzami

Showing results (31-40 of 75) with videos related to

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The Journal of Experimental Medicine|May 1, 1995
Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivoN Zamzami, P Marchetti, M Castedo, et al.
Clinical Immunology and Immunopathology|March 1, 1996
Polyethylene glycol-modified IL-2 abrogates superantigen-induced anergy without affecting peripheral clonal deletion in vivoA González-García, P Marchetti, M Castedo, et al.
The Journal of Experimental Medicine|April 1, 1996
Mitochondrial control of nuclear apoptosisN Zamzami, S A Susin, P Marchetti, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 30, 1998
Potassium leakage during the apoptotic degradation phaseB Dallaporta, T Hirsch, S A Susin, et al.
Oncogene|October 31, 1997
The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell deathT Hirsch, P Marchetti, S A Susin, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 1, 1998
Proteasome activation occurs at an early, premitochondrial step of thymocyte apoptosisT Hirsch, B Dallaporta, N Zamzami, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 1, 1996
Mitochondrial permeability transition triggers lymphocyte apoptosisP Marchetti, T Hirsch, N Zamzami, et al.
Oncogene|May 22, 2001
Endoplasmic reticulum localized Bcl-2 prevents apoptosis when redistribution of cytochrome c is a late eventM G Annis, N Zamzami, W Zhu, et al.
FEBS Letters|June 30, 1997
Nitric oxide induces apoptosis via triggering mitochondrial permeability transitionS Hortelano, B Dallaporta, N Zamzami, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 15, 1997
Glutathione depletion is an early and calcium elevation is a late event of thymocyte apoptosisA Macho, T Hirsch, I Marzo, et al.
Pageof 8

Showing results (31-40 of 75) with videos related to

Sort By:
Pageof 8
The Journal of Experimental Medicine|May 1, 1995
Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivoN Zamzami, P Marchetti, M Castedo, et al.
Clinical Immunology and Immunopathology|March 1, 1996
Polyethylene glycol-modified IL-2 abrogates superantigen-induced anergy without affecting peripheral clonal deletion in vivoA González-García, P Marchetti, M Castedo, et al.
The Journal of Experimental Medicine|April 1, 1996
Mitochondrial control of nuclear apoptosisN Zamzami, S A Susin, P Marchetti, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 30, 1998
Potassium leakage during the apoptotic degradation phaseB Dallaporta, T Hirsch, S A Susin, et al.
Oncogene|October 31, 1997
The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell deathT Hirsch, P Marchetti, S A Susin, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 1, 1998
Proteasome activation occurs at an early, premitochondrial step of thymocyte apoptosisT Hirsch, B Dallaporta, N Zamzami, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 1, 1996
Mitochondrial permeability transition triggers lymphocyte apoptosisP Marchetti, T Hirsch, N Zamzami, et al.
Oncogene|May 22, 2001
Endoplasmic reticulum localized Bcl-2 prevents apoptosis when redistribution of cytochrome c is a late eventM G Annis, N Zamzami, W Zhu, et al.
FEBS Letters|June 30, 1997
Nitric oxide induces apoptosis via triggering mitochondrial permeability transitionS Hortelano, B Dallaporta, N Zamzami, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 15, 1997
Glutathione depletion is an early and calcium elevation is a late event of thymocyte apoptosisA Macho, T Hirsch, I Marzo, et al.
Pageof 8