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Epilepsia|August 28, 2018
Transient microstructural brain anomalies and epileptiform discharges in mice defective for epilepsy and language-related NMDA receptor subunit gene Grin2aManal Salmi, Radu Bolbos, Sylvian Bauer, et al.
Neuron|May 25, 2007
Increased threshold for spike-timing-dependent plasticity is caused by unreliable calcium signaling in mice lacking fragile X gene FMR1Rhiannon M Meredith, Carl D Holmgren, Meredith Weidum, et al.
Frontiers in Cellular Neuroscience|August 18, 2011
Phenobarbital but Not Diazepam Reduces AMPA/kainate Receptor Mediated Currents and Exerts Opposite Actions on Initial Seizures in the Neonatal Rat HippocampusRomain Nardou, Sumii Yamamoto, Asma Bhar, et al.
Frontiers in Cellular Neuroscience|June 15, 2017
Functional Properties of Human NMDA Receptors Associated with Epilepsy-Related Mutations of GluN2A SubunitDmitry A Sibarov, Nadine Bruneau, Sergei M Antonov, et al.
The Journal of Physiology|October 2, 2004
NMDA receptors induce somatodendritic secretion in hypothalamic neurones of lactating female ratsChristiaan P J de Kock, Nail Burnashev, Johannes C Lodder, et al.
Cerebral Cortex (New York, N.Y. : 1991)|May 18, 2018
Term or Preterm Cesarean Section Delivery Does Not Lead to Long-term Detrimental Consequences in MiceMorgane Chiesa, Damien Guimond, Roman Tyzio, et al.
Epilepsia|May 8, 2024
Pathogenic MTOR somatic variant causing focal cortical dysplasia drives hyperexcitability via overactivation of neuronal GluN2C N-methyl-D-aspartate receptorsLouison Pineau, Emmanuelle Buhler, Sarah Tarhini, et al.
Science (New York, N.Y.)|October 11, 2014
Response to Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring"Sanaz Eftekhari, Amene Shahrokhi, Vera Tsintsadze, et al.
Cerebral Cortex (New York, N.Y. : 1991)|November 6, 2018
The GABA Developmental Shift Is Abolished by Maternal Immune Activation Already at BirthAmandine Fernandez, Camille Dumon, Damien Guimond, et al.
The European Journal of Neuroscience|April 25, 2006
Non-fibrillar beta-amyloid abates spike-timing-dependent synaptic potentiation at excitatory synapses in layer 2/3 of the neocortex by targeting postsynaptic AMPA receptorsIsaac Shemer, Carl Holmgren, Rogier Min, et al.
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