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Cell Biochemistry and Biophysics|April 28, 2010
MMP-9 gene ablation and TIMP-4 mitigate PAR-1-mediated cardiomyocyte dysfunction: a plausible role of dicer and miRNAParas Kumar Mishra, Naira Metreveli, Suresh C Tyagi
Plos One|January 4, 2014
Folic acid mitigates angiotensin-II-induced blood pressure and renal remodelingSathnur B Pushpakumar, Sourav Kundu, Naira Metreveli, et al.
Biochemical and Biophysical Research Communications|September 15, 2010
Attenuation of beta2-adrenergic receptors and homocysteine metabolic enzymes cause diabetic cardiomyopathyParas Kumar Mishra, Srikanth Givvimani, Naira Metreveli, et al.
Canadian Journal of Physiology and Pharmacology|March 8, 2012
Ablation of MMP9 induces survival and differentiation of cardiac stem cells into cardiomyocytes in the heart of diabetics: a role of extracellular matrixParas Kumar Mishra, Vishalakshi Chavali, Naira Metreveli, et al.
Current Vascular Pharmacology|April 9, 2008
Mitochondrial MMP activation, dysfunction and arrhythmogenesis in hyperhomocysteinemiaKarni S Moshal, Naira Metreveli, Iuliana Frank, et al.
American Journal of Physiology. Heart and Circulatory Physiology|November 22, 2011
Autophagy mechanism of right ventricular remodeling in murine model of pulmonary artery constrictionNatia Qipshidze, Neetu Tyagi, Naira Metreveli, et al.
Molecular Biology Reports|May 31, 2014
MMP-9 gene ablation mitigates hyperhomocystenemia-induced cognition and hearing dysfunctionSeema Bhargava, Sathnur Pushpakumar, Naira Metreveli, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|May 29, 2014
Ablation of MMP9 gene ameliorates paracellular permeability and fibrinogen-amyloid beta complex formation during hyperhomocysteinemiaNino Muradashvili, Reeta Tyagi, Naira Metreveli, et al.
Canadian Journal of Physiology and Pharmacology|June 3, 2015
A possible molecular mechanism of hearing loss during cerebral ischemia in micePradip Kumar Kamat, Anuradha Kalani, Naira Metreveli, et al.
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