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Journal of Cell Science|January 28, 2014
Mutation of caspase-digestion sites in keratin 18 interferes with filament reorganization, and predisposes to hepatocyte necrosis and loss of membrane integritySujith V W Weerasinghe, Nam-On Ku, Peter J Altshuler, et al.Human Mutation|April 1, 2006
Denaturing temperature selection may underestimate keratin mutation detection by DHPLCPavel Strnad, Tim Christian Lienau, Guo-Zhong Tao, et al.Journal of Cell Science|August 3, 2021
Keratin 8 mutations in transgenic mice predispose to lung injurySujin Kim, Younglan Lim, So-Young Lee, et al.Hepatology (Baltimore, Md.)|September 16, 2004
Keratin 8 and 18 hyperphosphorylation is a marker of progression of human liver diseaseDiana M Toivola, Nam-On Ku, Evelyn Z Resurreccion, et al.Plos One|November 5, 2010
Characterization of in vivo keratin 19 phosphorylation on tyrosine-391Qin Zhou, Natasha T Snider, Jian Liao, et al.Experimental Cell Research|December 14, 2005
Bispecific and human disease-related anti-keratin rabbit monoclonal antibodiesGuo-Zhong Tao, Ikuo Nakamichi, Nam-On Ku, et al.Biology Open|July 18, 2013
Predisposition to apoptosis in keratin 8-null liver is related to inactivation of NF-κB and SAPKs but not decreased c-FlipJongeun Lee, Kwi-Hoon Jang, Hakhyun Kim, et al.Gastroenterology|September 7, 2005
Keratins as susceptibility genes for end-stage liver diseaseNam-On Ku, Joseph K Lim, Sheri M Krams, et al.Proceedings of the National Academy of Sciences of the United States of America|May 2, 2003
Keratin 8 and 18 mutations are risk factors for developing liver disease of multiple etiologiesNam-On Ku, Jama M Darling, Sheri M Krams, et al.Cell Reports|April 28, 2024
O-GlcNAcylation of RBM14 contributes to elevated cellular O-GlcNAc through regulation of OGA protein stabilityTae Hyun Kweon, Hyeryeon Jung, Jeong Yeon Ko, et al.Pageof 4