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Molecular Carcinogenesis|September 16, 2005
Suppression of p65 phosphorylation coincides with inhibition of IkappaBalpha polyubiquitination and degradationJing Hu, Mohammed Haseebuddin, Matthew Young, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 6, 2008
Sulfiredoxin is an AP-1 target gene that is required for transformation and shows elevated expression in human skin malignanciesQiou Wei, Hong Jiang, Connie P Matthews, et al.
Oncogene|June 13, 2003
Pdcd4 suppresses tumor phenotype in JB6 cells by inhibiting AP-1 transactivationHsin-Sheng Yang, Jennifer L Knies, Cristi Stark, et al.
Molecular Cancer Therapeutics|February 27, 2004
Characterization of programmed cell death 4 in multiple human cancers reveals a novel enhancer of drug sensitivityAaron P Jansen, Corinne E Camalier, Cristi Stark, et al.
Experimental Cell Research|February 22, 2002
Activation of NF-kappaB is required for PDGF-B chain to transform NIH3T3 cellsTakeshi Shimamura, Tin-Chen Hsu, Nancy H Colburn, et al.
The Journal of Nutritional Biochemistry|June 19, 2012
Cancer stem cells: potential target for bioactive food componentsYoung S Kim, William Farrar, Nancy H Colburn, et al.
Breast Cancer Research and Treatment|April 4, 2008
Programmed cell death 4 inhibits breast cancer cell invasion by increasing tissue inhibitor of metalloproteinases-2 expressionRené Nieves-Alicea, Nancy H Colburn, Ann-Marie Simeone, et al.
Neuro-Oncology|June 4, 2011
Downregulation of Pdcd4 by mir-21 facilitates glioblastoma proliferation in vivoArti B Gaur, Susan L Holbeck, Nancy H Colburn, et al.
Carcinogenesis|January 14, 2014
Tumor suppressor PDCD4 inhibits NF-κB-dependent transcription in human glioblastoma cells by direct interaction with p65Soon-Kyung Hwang, Alyson R Baker, Matthew R Young, et al.
Molecular Carcinogenesis|July 12, 2002
Protection against human papillomavirus type 16-E7 oncogene-induced tumorigenesis by in vivo expression of dominant-negative c-junMatthew R Young, Linda Farrell, Paul Lambert, et al.
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