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Antioxidants (Basel, Switzerland)|July 27, 2024
Ocular Inflammation and Oxidative Stress as a Result of Chronic Intermittent Hypoxia: A Rat Model of Sleep ApneaNina Donkor, Jennifer J Gardner, Jessica L Bradshaw, et al.
Investigative Ophthalmology & Visual Science|March 1, 2006
Microarray analysis of retinal gene expression in the DBA/2J model of glaucomaMichael R Steele, Denise M Inman, David J Calkins, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 3, 2010
Distal axonopathy with structural persistence in glaucomatous neurodegenerationSamuel D Crish, Rebecca M Sappington, Denise M Inman, et al.
Experimental Eye Research|December 10, 2015
Early astrocyte redistribution in the optic nerve precedes axonopathy in the DBA/2J mouse model of glaucomaMelissa L Cooper, Samuel D Crish, Denise M Inman, et al.
Journal of Neuroscience Research|January 27, 2004
Treatment with the neurosteroid dehydroepiandrosterone promotes recovery of motor behavior after moderate contusive spinal cord injury in the mouseChristelle Fiore, Denise M Inman, Shijiro Hirose, et al.
Frontiers in Neuroscience|November 19, 2016
Early Cytoskeletal Protein Modifications Precede Overt Structural Degeneration in the DBA/2J Mouse Model of GlaucomaGina N Wilson, Matthew A Smith, Denise M Inman, et al.
Neurobiology of Disease|May 19, 2020
MCT2 overexpression rescues metabolic vulnerability and protects retinal ganglion cells in two models of glaucomaMohammad Harun-Or-Rashid, Nathaniel Pappenhagen, Ryan Zubricky, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 1, 2013
Differential progression of structural and functional alterations in distinct retinal ganglion cell types in a mouse model of glaucomaLuca Della Santina, Denise M Inman, Caroline B Lupien, et al.
Experimental Neurology|May 19, 2009
Drp1 levels constitutively regulate mitochondrial dynamics and cell survival in cortical neuronsTakuma Uo, Jenny Dworzak, Chizuru Kinoshita, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 23, 2010
Metabolic vulnerability disposes retinal ganglion cell axons to dysfunction in a model of glaucomatous degenerationSelva Baltan, Denise M Inman, Camelia A Danilov, et al.
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