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Blood|September 2, 2009
Colony-stimulating factor-1-induced oscillations in phosphatidylinositol-3 kinase/AKT are required for caspase activation in monocytes undergoing differentiation into macrophagesArnaud Jacquel, Naïma Benikhlef, Jérôme Paggetti, et al.
Cell Biology International|June 14, 2008
Tumor cells can escape DNA-damaging cisplatin through DNA endoreduplication and reversible polyploidyPierre-Emmanuel Puig, Marie-Noëlle Guilly, André Bouchot, et al.
Blood Advances|January 4, 2018
Engraftment of chronic myelomonocytic leukemia cells in immunocompromised mice supports disease dependency on cytokinesYanyan Zhang, Liang He, Dorothée Selimoglu-Buet, et al.
Oncoimmunology|March 7, 2022
Reprogramming monocyte-derived macrophages through caspase inhibitionPaul Chaintreuil, Lucie Laplane, Florian Esnault, et al.
Blood|August 31, 2011
Fine-tuning nucleophosmin in macrophage differentiation and activationLeslie Guery, Naïma Benikhlef, Thomas Gautier, et al.
Cancer Research|April 17, 2004
Increased immunogenicity of colon cancer cells by selective depletion of cytochrome CElise Schmitt, Arnaud Parcellier, François Ghiringhelli, et al.
Nature Communications|April 28, 2019
Dynamic gene regulation by nuclear colony-stimulating factor 1 receptor in human monocytes and macrophagesLaura Bencheikh, M'Boyba Khadija Diop, Julie Rivière, et al.
Critical Care Medicine|June 20, 2014
Circulating immature granulocytes with T-cell killing functions predict sepsis deterioration*Estelle Guérin, Marie Orabona, Marie-Astrid Raquil, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 16, 2006
Cytoprotective gene bi-1 is required for intrinsic protection from endoplasmic reticulum stress and ischemia-reperfusion injuryBéatrice Bailly-Maitre, Constantino Fondevila, Fady Kaldas, et al.
The Journal of Clinical Investigation|May 4, 2011
Transcription intermediary factor 1γ is a tumor suppressor in mouse and human chronic myelomonocytic leukemiaRomain Aucagne, Nathalie Droin, Jérôme Paggetti, et al.
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