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DNA and Cell Biology|March 27, 2014
The innate immune response to HIV-1: to sense or not to senseNathaniel R LandauTrends in Pharmacological Sciences|June 27, 2002
A cellular cofactor in HIV-1 assembly: INI1 is also an "outtie"Nathaniel R LandauCurrent Protocols in Pharmacology|September 30, 2011
Production and use of HIV-1 luciferase reporter virusesCarsten Münk, Nathaniel R LandauThe Journal of Biological Chemistry|October 6, 2006
Reversed functional organization of mouse and human APOBEC3 cytidine deaminase domainsYoshiyuki Hakata, Nathaniel R LandauScientific Reports|March 9, 2018
DNA damage induces a SAMHD1-mediated block to the infection of macrophages by HIV-1Paula Jáuregui, Nathaniel R LandauVirology|October 20, 2006
Analysis of Vif-induced APOBEC3G degradation using an alpha-complementation assayLei Fang, Nathaniel R LandauCurrent Opinion in Immunology|July 13, 2004
Recent insights into HIV-1 VifFrancisco Navarro, Nathaniel R LandauVirology Journal|June 11, 2014
Interactions with DCAF1 and DDB1 in the CRL4 E3 ubiquitin ligase are required for Vpr-mediated G2 arrestYoshiyuki Hakata, Masaaki Miyazawa, Nathaniel R LandauNature Immunology|May 20, 2015
Intrinsic host restrictions to HIV-1 and mechanisms of viral escapeViviana Simon, Nicolin Bloch, Nathaniel R LandauProceedings of the National Academy of Sciences of the United States of America|February 24, 2004
A single amino acid of APOBEC3G controls its species-specific interaction with virion infectivity factor (Vif)Bärbel Schröfelbauer, Darlene Chen, Nathaniel R LandauPageof 9