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Free Radical Biology & Medicine|December 27, 2006
The cellular basis for diverse responses to oxygenNavdeep S Chandel, G R Scott BudingerGenes & Development|July 11, 2025
Mitochondria dysfunction: cause or consequence of physiologic aging?G R Scott Budinger, Navdeep S ChandelScience Translational Medicine|July 5, 2013
The good and the bad of antibioticsNavdeep S Chandel, G R Scott BudingerAmerican Journal of Physiology. Lung Cellular and Molecular Physiology|October 12, 2002
Hypoxic but not anoxic stabilization of HIF-1alpha requires mitochondrial reactive oxygen speciesClara Schroedl, David S McClintock, G R Scott Budinger, et al.Translational Research : the Journal of Laboratory and Clinical Medicine|October 29, 2017
Reactive oxygen species as signaling molecules in the development of lung fibrosisFrancisco J Gonzalez-Gonzalez, Navdeep S Chandel, Manu Jain, et al.The Journal of Biological Chemistry|November 20, 2003
Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cellsLeonard J Buccellato, May Tso, Ozkan I Akinci, et al.Intensive Care Medicine|November 27, 2004
Active transforming growth factor-beta1 activates the procollagen I promoter in patients with acute lung injuryG R Scott Budinger, Navdeep S Chandel, Helen K Donnelly, et al.The Journal of Biological Chemistry|February 28, 2002
Hypoxia sensitizes cells to nitric oxide-induced apoptosisVivian Y Lee, David S McClintock, Matthew T Santore, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|March 7, 2002
Anoxia-induced apoptosis occurs through a mitochondria-dependent pathway in lung epithelial cellsMatthew T Santore, David S McClintock, Vivian Y Lee, et al.The Journal of Biological Chemistry|March 6, 2002
Hyperoxia-induced apoptosis does not require mitochondrial reactive oxygen species and is regulated by Bcl-2 proteinsG R Scott Budinger, May Tso, David S McClintock, et al.Pageof 43