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Plos One|May 16, 2014
High molecular weight fibroblast growth factor-2 in the human heart is a potential target for prevention of cardiac remodelingJon-Jon Santiago, Leslie J McNaughton, Navid Koleini, et al.
Biorxiv : the Preprint Server for Biology|June 25, 2024
ATP Citrate Lyase Supports Cardiac Function and NAD+/NADH Balance And Is Depressed in Human Heart FailureMariam Meddeb, Navid Koleini, Seungho Jun, et al.
Biorxiv : the Preprint Server for Biology|June 12, 2026
Reduced Myocardial Serine Synthesis Impairs Functional, Metabolic, and Redox Adaptations to Cardiac StressMalihe Rezaee, Mohammad Keykhaei, Navid Koleini, et al.
Circulation|March 1, 2023
Myocardial Metabolomics of Human Heart Failure With Preserved Ejection FractionVirginia S Hahn, Christopher Petucci, Min-Soo Kim, et al.
Cell and Tissue Research|May 31, 2021
Elimination of endogenous high molecular weight FGF2 prevents pressure-overload-induced systolic dysfunction, linked to increased FGFR1 activity and NR1D1 expressionNavid Koleini, Barbara E Nickel, Raghu S Nagalingam, et al.
JCI Insight|October 3, 2023
TSC2 S1365A mutation potently regulates CD8+ T cell function and differentiation and improves adoptive cellular cancer therapyChirag H Patel, Yi Dong, Navid Koleini, et al.
JACC. Basic to Translational Science|June 11, 2025
ATP-Citrate Lyase Supports Cardiac Function and NAD<sup>+</sup>/NADH Balance and Is Depressed in Human Failing MyocardiumMariam Meddeb, Navid Koleini, Mohammad Keykhaei, et al.
Basic Research in Cardiology|April 13, 2021
SKI activates the Hippo pathway via LIMD1 to inhibit cardiac fibroblast activationNatalie M Landry, Sunil G Rattan, Krista L Filomeno, et al.
Nature Cardiovascular Research|August 28, 2024
Myocardial ultrastructure of human heart failure with preserved ejection fractionMariam Meddeb, Navid Koleini, Aleksandra Binek, et al.
The FEBS Journal|September 24, 2019
Simvastatin increases temozolomide-induced cell death by targeting the fusion of autophagosomes and lysosomesShahla Shojaei, Navid Koleini, Ehsan Samiei, et al.
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