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International Journal of Molecular Sciences
|
October 7, 2020
The Effects of Immune System Modulation on Prion Disease Susceptibility and Pathogenesis
Neil A Mabbott, Barry M Bradford, Reiss Pal, et al.
Plos Pathogens
|
December 16, 2016
Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility
David S Donaldson, Anuj Sehgal, Daniel Rios, et al.
Scientific Reports
|
June 14, 2024
Cell adhesion molecule CD44 is dispensable for reactive astrocyte activation during prion disease
Barry M Bradford, Lauryn Walmsley-Rowe, Joe Reynolds, et al.
Journal of Virology
|
July 20, 2018
Oral Prion Neuroinvasion Occurs Independently of PrP<sup>C</sup> Expression in the Gut Epithelium
Alison Marshall, Barry M Bradford, Alan R Clarke, et al.
Glia
|
July 19, 2022
Microglia deficiency accelerates prion disease but does not enhance prion accumulation in the brain
Barry M Bradford, Lynne I McGuire, David A Hume, et al.
Journal of Dermatological Science
|
July 22, 2004
Scrapie transmission following exposure through the skin is dependent on follicular dendritic cells in lymphoid tissues
Joanne Mohan, Karen L Brown, Christine F Farquhar, et al.
Scientific Reports
|
May 2, 2019
Effect of co-infection with a small intestine-restricted helminth pathogen on oral prion disease pathogenesis in mice
Alejandra Sánchez-Quintero, Barry M Bradford, Rick Maizels, et al.
BMC Genomics
|
September 24, 2013
An expression atlas of human primary cells: inference of gene function from coexpression networks
Neil A Mabbott, J Kenneth Baillie, Helen Brown, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
September 30, 2009
The effects of host age on follicular dendritic cell status dramatically impair scrapie agent neuroinvasion in aged mice
Karen L Brown, Gwennaelle J Wathne, Jill Sales, et al.
Parasite Immunology
|
June 20, 2018
Increased susceptibility to oral Trichuris muris infection in the specific absence of CXCR5<sup>+</sup> CD11c<sup>+</sup> cells
Barry M Bradford, David S Donaldson, Ruth Forman, et al.
Page
of 13
Search research articles
Search
Showing results (61-70 of 123) with videos related to
Sort By:
Page
of 13
International Journal of Molecular Sciences
|
October 7, 2020
The Effects of Immune System Modulation on Prion Disease Susceptibility and Pathogenesis
Neil A Mabbott, Barry M Bradford, Reiss Pal, et al.
Plos Pathogens
|
December 16, 2016
Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility
David S Donaldson, Anuj Sehgal, Daniel Rios, et al.
Scientific Reports
|
June 14, 2024
Cell adhesion molecule CD44 is dispensable for reactive astrocyte activation during prion disease
Barry M Bradford, Lauryn Walmsley-Rowe, Joe Reynolds, et al.
Journal of Virology
|
July 20, 2018
Oral Prion Neuroinvasion Occurs Independently of PrP<sup>C</sup> Expression in the Gut Epithelium
Alison Marshall, Barry M Bradford, Alan R Clarke, et al.
Glia
|
July 19, 2022
Microglia deficiency accelerates prion disease but does not enhance prion accumulation in the brain
Barry M Bradford, Lynne I McGuire, David A Hume, et al.
Journal of Dermatological Science
|
July 22, 2004
Scrapie transmission following exposure through the skin is dependent on follicular dendritic cells in lymphoid tissues
Joanne Mohan, Karen L Brown, Christine F Farquhar, et al.
Scientific Reports
|
May 2, 2019
Effect of co-infection with a small intestine-restricted helminth pathogen on oral prion disease pathogenesis in mice
Alejandra Sánchez-Quintero, Barry M Bradford, Rick Maizels, et al.
BMC Genomics
|
September 24, 2013
An expression atlas of human primary cells: inference of gene function from coexpression networks
Neil A Mabbott, J Kenneth Baillie, Helen Brown, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
September 30, 2009
The effects of host age on follicular dendritic cell status dramatically impair scrapie agent neuroinvasion in aged mice
Karen L Brown, Gwennaelle J Wathne, Jill Sales, et al.
Parasite Immunology
|
June 20, 2018
Increased susceptibility to oral Trichuris muris infection in the specific absence of CXCR5<sup>+</sup> CD11c<sup>+</sup> cells
Barry M Bradford, David S Donaldson, Ruth Forman, et al.
Page
of 13