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JCI Insight|September 8, 2023
Structural and functional analyses of a germline KRAS T50I mutation provide insights into Raf activationPan-Yu Chen, Benjamin J Huang, Max Harris, et al.Human Mutation|November 6, 2007
Mutation analysis in Costello syndrome: functional and structural characterization of the HRAS p.Lys117Arg mutationEllen Denayer, Annabel Parret, Magdalena Chmara, et al.Proceedings of the National Academy of Sciences of the United States of America|March 14, 2014
Crenolanib is a selective type I pan-FLT3 inhibitorCatherine Choy Smith, Elisabeth A Lasater, Kimberly C Lin, et al.Biorxiv : the Preprint Server for Biology|November 26, 2025
Acute Myeloid Leukemia Relapse after Bromodomain Inhibitor Treatment or Chemotherapy is Characterized by Myc-Ras Transcriptional RemodelingBenjamin J Huang, Jeevitha D'Souza, Alexa Rane Batingana, et al.Blood|May 12, 2017
Heterogeneous resistance to quizartinib in acute myeloid leukemia revealed by single-cell analysisCatherine C Smith, Amy Paguirigan, Grace R Jeschke, et al.Cancer Discovery|June 5, 2013
Dominant role of oncogene dosage and absence of tumor suppressor activity in Nras-driven hematopoietic transformationJin Xu, Kevin M Haigis, Ari J Firestone, et al.Cardiology in the Young|June 8, 2022
Transseptal puncture during catheter ablation associated with higher radiation exposureMaryam Rahman, Grace Smith, Chris Johnsrude, et al.Leukemia & Lymphoma|October 25, 2019
Dasatinib discontinuation in patients with chronic-phase chronic myeloid leukemia and stable deep molecular response: the DASFREE studyNeil P Shah, Valentín García-Gutiérrez, Antonio Jiménez-Velasco, et al.Haematologica|May 5, 2022
Patient- and physician-reported pain after tyrosine kinase inhibitor discontinuation among patients with chronic myeloid leukemiaKathryn E Flynn, Ehab Atallah, Li Lin, et al.Nature|April 17, 2012
Validation of ITD mutations in FLT3 as a therapeutic target in human acute myeloid leukaemiaCatherine C Smith, Qi Wang, Chen-Shan Chin, et al.Pageof 21