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RSC Chemical Biology|August 30, 2021
Correction: In vivo modulation of ubiquitin chains by N-methylated non-proteinogenic cyclic peptidesJoseph M Rogers, Mickal Nawatha, Betsegaw Lemma, et al.BMC Cancer|April 1, 2017
Identification of Novel Class of Triazolo-Thiadiazoles as Potent Inhibitors of Human Heparanase and their Anticancer ActivityC P Baburajeev, Chakrabhavi Dhananjaya Mohan, Shobith Rangappa, et al.Diabetes|March 11, 2014
Endothelial cell heparanase taken up by cardiomyocytes regulates lipoprotein lipase transfer to the coronary lumen after diabetesYing Wang, Amy Pei-Ling Chiu, Katharina Neumaier, et al.Journal of the American Heart Association|November 23, 2022
Flow-Induced Secretion of Endothelial Heparanase Regulates Cardiac Lipoprotein Lipase and Changes Following DiabetesChae Syng Lee, Yajie Zhai, Rui Shang, et al.Cancer Letters|November 8, 2005
A new targeting approach for breast cancer gene therapy using the heparanase promoterMartina Breidenbach, Daniel T Rein, Thomas Schöndorf, et al.Development (Cambridge, England)|October 26, 2007
Heparanase cleavage of perlecan heparan sulfate modulates FGF10 activity during ex vivo submandibular gland branching morphogenesisVaishali N Patel, Sarah M Knox, Karen M Likar, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|October 12, 2013
Hyperglycemia-induced secretion of endothelial heparanase stimulates a vascular endothelial growth factor autocrine network in cardiomyocytes that promotes recruitment of lipoprotein lipaseDahai Zhang, Andrea Wan, Amy Pei-Ling Chiu, et al.Bioorganic & Medicinal Chemistry|January 1, 2025
Synthetic glycol-split heparin tri- and tetrasaccharides provide new insights into structural peculiarities for antiheparanase activityMinghong Ni, Michela Parafioriti, Emiliano Esposito, et al.The Journal of Biological Chemistry|March 10, 2007
Heparanase enhances syndecan-1 shedding: a novel mechanism for stimulation of tumor growth and metastasisYang Yang, Veronica Macleod, Hua-Quan Miao, et al.Cell Death & Disease|October 21, 2018
AMPK-dependent autophagy upregulation serves as a survival mechanism in response to Tumor Treating Fields (TTFields)Anna Shteingauz, Yaara Porat, Tali Voloshin, et al.Pageof 31