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Plos Pathogens|March 23, 2022
Human papillomavirus type 16 E6 induces cell competitionNicole Brimer, Scott Vande PolJournal of Virology|December 20, 2013
Papillomavirus E6 PDZ interactions can be replaced by repression of p53 to promote episomal human papillomavirus genome maintenanceNicole Brimer, Scott B Vande PolJournal of Virology|April 4, 2008
Transformation by bovine papillomavirus type 1 E6 requires paxillinRamon Wade, Nicole Brimer, Scott Vande PolJournal of Virology|June 20, 2014
Interactions between E6, FAK, and GIT1 at paxillin LD4 are necessary for transformation by bovine papillomavirus 1 E6Nicole Brimer, Ramon Wade, Scott Vande PolVirology|October 7, 2006
Association of E6AP (UBE3A) with human papillomavirus type 11 E6 proteinNicole Brimer, Charles Lyons, Scott B Vande PolJournal of Virology|August 17, 2012
Peptide interactions stabilize and restructure human papillomavirus type 16 E6 to interact with p53Tina Ansari, Nicole Brimer, Scott B Vande PolJournal of Virology|September 7, 2007
Human papillomavirus E6 regulates the cytoskeleton dynamics of keratinocytes through targeted degradation of p53Brooke Cooper, Nicole Brimer, Scott B Vande PolPlos Pathogens|January 24, 2020
Multiple regions of E6AP (UBE3A) contribute to interaction with papillomavirus E6 proteins and the activation of ubiquitin ligase activityCamille M Drews, Nicole Brimer, Scott B Vande PolPlos Pathogens|December 28, 2017
Association of papillomavirus E6 proteins with either MAML1 or E6AP clusters E6 proteins by structure, function, and evolutionary relatednessNicole Brimer, Camille M Drews, Scott B Vande PolThe Journal of Biological Chemistry|September 9, 2011
Paxillin enables attachment-independent tyrosine phosphorylation of focal adhesion kinase and transformation by RASRamon Wade, Nicole Brimer, Charles Lyons, et al.Pageof 2