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The Journal of Biological Chemistry|July 25, 2022
CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticitySarah G Cook, Nicole L Rumian, K Ulrich Bayer
Stem Cell Reports|May 29, 2018
Retinoic Acid Is Required for Neural Stem and Progenitor Cell Proliferation in the Adult HippocampusSwati Mishra, Kathleen K Kelly, Nicole L Rumian, et al.
Current Opinion in Neurobiology|March 31, 2025
CaMKII mechanisms that promote pathological LTP impairmentsMatthew E Larsen, Nicole L Rumian, Nidia Quillinan, et al.
Plos One|July 28, 2020
The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targetingJonathan E Tullis, Nicole L Rumian, Carolyn Nicole Brown, et al.
Science Signaling|July 25, 2023
Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in miceNicole L Rumian, Ronald K Freund, Mark L Dell'Acqua, et al.
Iscience|May 27, 2022
Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling eventsCarolyn Nicole Brown, Nicole L Rumian, Jonathan E Tullis, et al.
Brain Research|October 23, 2021
CaMKIIα knockout protects from ischemic neuronal cell death after resuscitation from cardiac arrestNicole L Rumian, Nicholas E Chalmers, Jonathan E Tullis, et al.
Molecular Biology of the Cell|March 26, 2025
Aβ impairs the LTP-related movement of endogenous CaMKII but not of exogenous GFP-CaMKIICarolyn Nicole Brown, Fan-Yi Chao, Daphne Quang, et al.
Cell Reports|October 12, 2024
LTP expression mediated by autonomous activity of GluN2B-bound CaMKIINicole L Rumian, C Madison Barker, Matthew E Larsen, et al.
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