Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Nils Brose

Showing results (41-50 of 228) with videos related to

Pageof 23
Sort By:
The Journal of Experimental Medicine|May 5, 2018
Autism-associated neuroligin-4 mutation selectively impairs glycinergic synaptic transmission in mouse brainstem synapsesBo Zhang, Ozgun Gokce, W Dylan Hale, et al.
The European Journal of Neuroscience|June 30, 2009
A common molecular basis for membrane docking and functional priming of synaptic vesiclesLéa Siksou, Frédérique Varoqueaux, Olivier Pascual, et al.
Cell Reports|March 7, 2019
Acute Complexin Knockout Abates Spontaneous and Evoked Transmitter ReleaseFrancisco José López-Murcia, Kerstin Reim, Olaf Jahn, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 29, 2015
Complexin stabilizes newly primed synaptic vesicles and prevents their premature fusion at the mouse calyx of held synapseShuwen Chang, Kerstin Reim, Meike Pedersen, et al.
The European Journal of Neuroscience|October 4, 2005
A similar impairment in CA3 mossy fibre LTP in the R6/2 mouse model of Huntington's disease and in the complexin II knockout mouseHelen E Gibson, Kerstin Reim, Nils Brose, et al.
Molecular Biology and Evolution|February 4, 2020
Evolution of the Autism-Associated Neuroligin-4 Gene Reveals Broad Erosion of Pseudoautosomal Regions in RodentsStephan Maxeiner, Fritz Benseler, Gabriela Krasteva-Christ, et al.
Behavioural Brain Research|May 24, 2014
Juvenile manifestation of ultrasound communication deficits in the neuroligin-4 null mutant mouse model of autismAnes Ju, Kurt Hammerschmidt, Martesa Tantra, et al.
Human Molecular Genetics|July 8, 2005
Profound ataxia in complexin I knockout mice masks a complex phenotype that includes exploratory and habituation deficitsDervila Glynn, Cheney J Drew, Kerstin Reim, et al.
Current Opinion in Neurobiology|December 30, 2016
Synapse biology in the 'circuit-age'-paths toward molecular connectomicsDietmar Schreiner, Jeffrey N Savas, Etienne Herzog, et al.
Science Advances|April 8, 2026
Selective, genetically induced increase in synaptic vesicle primingMohammad Aldahabi, Flora Balint, Andrea Lorincz, et al.
Pageof 23

Showing results (41-50 of 228) with videos related to

Sort By:
Pageof 23
The Journal of Experimental Medicine|May 5, 2018
Autism-associated neuroligin-4 mutation selectively impairs glycinergic synaptic transmission in mouse brainstem synapsesBo Zhang, Ozgun Gokce, W Dylan Hale, et al.
The European Journal of Neuroscience|June 30, 2009
A common molecular basis for membrane docking and functional priming of synaptic vesiclesLéa Siksou, Frédérique Varoqueaux, Olivier Pascual, et al.
Cell Reports|March 7, 2019
Acute Complexin Knockout Abates Spontaneous and Evoked Transmitter ReleaseFrancisco José López-Murcia, Kerstin Reim, Olaf Jahn, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 29, 2015
Complexin stabilizes newly primed synaptic vesicles and prevents their premature fusion at the mouse calyx of held synapseShuwen Chang, Kerstin Reim, Meike Pedersen, et al.
The European Journal of Neuroscience|October 4, 2005
A similar impairment in CA3 mossy fibre LTP in the R6/2 mouse model of Huntington's disease and in the complexin II knockout mouseHelen E Gibson, Kerstin Reim, Nils Brose, et al.
Molecular Biology and Evolution|February 4, 2020
Evolution of the Autism-Associated Neuroligin-4 Gene Reveals Broad Erosion of Pseudoautosomal Regions in RodentsStephan Maxeiner, Fritz Benseler, Gabriela Krasteva-Christ, et al.
Behavioural Brain Research|May 24, 2014
Juvenile manifestation of ultrasound communication deficits in the neuroligin-4 null mutant mouse model of autismAnes Ju, Kurt Hammerschmidt, Martesa Tantra, et al.
Human Molecular Genetics|July 8, 2005
Profound ataxia in complexin I knockout mice masks a complex phenotype that includes exploratory and habituation deficitsDervila Glynn, Cheney J Drew, Kerstin Reim, et al.
Current Opinion in Neurobiology|December 30, 2016
Synapse biology in the 'circuit-age'-paths toward molecular connectomicsDietmar Schreiner, Jeffrey N Savas, Etienne Herzog, et al.
Science Advances|April 8, 2026
Selective, genetically induced increase in synaptic vesicle primingMohammad Aldahabi, Flora Balint, Andrea Lorincz, et al.
Pageof 23