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Frontiers in Physiology|August 28, 2014
Reactive oxygen species and redox compartmentalizationNina Kaludercic, Soni Deshwal, Fabio Di LisaMethods in Molecular Biology (Clifton, N.J.)|June 1, 2018
Measurement of Mitochondrial ROS FormationSoni Deshwal, Salvatore Antonucci, Nina Kaludercic, et al.Current Opinion in Pharmacology|May 22, 2017
Emerging role of monoamine oxidase as a therapeutic target for cardiovascular diseaseSoni Deshwal, Moises Di Sante, Fabio Di Lisa, et al.The Journal of Biological Chemistry|November 28, 2020
The energetic cost of NNT-dependent ROS removalNina Kaludercic, Fabio Di LisaFrontiers in Cardiovascular Medicine|March 6, 2020
Mitochondrial ROS Formation in the Pathogenesis of Diabetic CardiomyopathyNina Kaludercic, Fabio Di LisaBritish Journal of Pharmacology|January 29, 2011
β₂-Adrenoceptors, NADPH oxidase, ROS and p38 MAPK: another 'radical' road to heart failure?Fabio Di Lisa, Nina Kaludercic, Nazareno PaolocciCell Calcium|January 30, 2023
IP3 receptor trafficking at the ER-mitochondria contacts impacts on mitochondrial Ca2+ homeostasis and metabolismKarim Rahhali, Fabio Di Lisa, Nina KaludercicCell Calcium|February 8, 2021
Mitochondrial reactive oxygen species in physiology and diseaseSalvatore Antonucci, Fabio Di Lisa, Nina KaludercicBasic Research in Cardiology|October 4, 2023
Recent advances on the role of monoamine oxidases in cardiac pathophysiologyNina Kaludercic, Ruth Jepchirchir Arusei, Fabio Di LisaBasic Research in Cardiology|January 20, 2023
Monoamine oxidase A-dependent ROS formation modulates human cardiomyocyte differentiation through AKT and WNT activationMoises Di Sante, Salvatore Antonucci, Laura Pontarollo, et al.Pageof 12