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Viruses|August 29, 2024
Role of PQBP1 in Pathogen Recognition-Impact on Innate ImmunityLukas Wiench, Domenico Rizzo, Zora Sinay, et al.
Journal of Virology|January 21, 2011
Expression of the human endogenous retrovirus (HERV) group HML-2/HERV-K does not depend on canonical promoter elements but is regulated by transcription factors Sp1 and Sp3Nina V Fuchs, Martin Kraft, Christiane Tondera, et al.
Retrovirology|October 26, 2013
Human endogenous retrovirus K (HML-2) RNA and protein expression is a marker for human embryonic and induced pluripotent stem cellsNina V Fuchs, Sabine Loewer, George Q Daley, et al.
The Journal of Biological Chemistry|December 3, 2016
Interferons Induce Expression of SAMHD1 in Monocytes through Down-regulation of miR-181a and miR-30aMaximilian Riess, Nina V Fuchs, Adam Idica, et al.
Mobile DNA|March 23, 2019
The impact of transposable element activity on therapeutically relevant human stem cellsGerald G Schumann, Nina V Fuchs, Pablo Tristán-Ramos, et al.
Journal of Virology|June 15, 2012
Biochemical analysis of the complex between the tetrameric export adapter protein Rec of HERV-K/HML-2 and the responsive RNA element RcRE pck30Janina S Langner, Nina V Fuchs, Jan Hoffmann, et al.
Stem Cell Research|December 15, 2019
Induced pluripotent stem cell line (PEIi003-A) derived from an apparently healthy male individualNina V Fuchs, Maximilian Schieck, Michaela Neuenkirch, et al.
Stem Cell Research|February 17, 2020
Generation of three induced pluripotent cell lines (iPSCs) from an Aicardi-Goutières syndrome (AGS) patient harboring a deletion in the genomic locus of the sterile alpha motif and HD domain containing protein 1 (SAMHD1)Nina V Fuchs, Maximilian Schieck, Michaela Neuenkirch, et al.
Stem Cell Research|November 8, 2019
Induced pluripotent stem cells (iPSCs) derived from a renpenning syndrome patient with c.459_462delAGAG mutation in PQBP1 (PEIi001-A)Nina V Fuchs, Maximilian Schieck, Michaela Neuenkirch, et al.
Nature Communications|June 10, 2018
Dephosphorylation of the HIV-1 restriction factor SAMHD1 is mediated by PP2A-B55α holoenzymes during mitotic exitKerstin Schott, Nina V Fuchs, Rita Derua, et al.
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