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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|October 5, 2018
T-lymphocyte-specific knockout of IKK-2 or NEMO induces T<sub>h</sub>17 cells in an experimental nephrotoxic nephritis mouse modelLinlin Guo, Jiabin Huang, Meilan Chen, et al.Bundesgesundheitsblatt, Gesundheitsforschung, Gesundheitsschutz|March 21, 2022
[Long-term physical and psychological consequences of chronic kidney disease]Friedrich ThaissAtherosclerosis. Supplements|February 5, 2010
Specific issues in living donor kidney transplantation: ABO - incompatibilityFriedrich ThaissCellular and Molecular Life Sciences : CMLS|April 19, 2023
IKK1 aggravates ischemia-reperfusion kidney injury by promoting the differentiation of effector T cellsNing Song, Yang Xu, Hans-Joachim Paust, et al.American Journal of Physiology. Renal Physiology|September 2, 2016
Lymphocyte-specific deletion of IKK2 or NEMO mediates an increase in intrarenal Th17 cells and accelerates renal damage in an ischemia-reperfusion injury mouse modelLinlin Guo, Hannah Heejung Lee, María de Las Mercedes Noriega, et al.Journal of Immunology Research|July 6, 2017
Innate Immune Response in Kidney Ischemia/Reperfusion Injury: Potential Target for TherapyAleksandra Kezić, Natasa Stajic, Friedrich ThaissTransplant International : Official Journal of the European Society for Organ Transplantation|September 25, 2007
Immunoadsorbtion and rituximab therapy in a second living-related kidney transplant patient with recurrent focal segmental glomerulosclerosisTobias N Meyer, Friedrich Thaiss, Rolf A K StahlHuman Immunology|January 6, 2019
Donor derived HLA-G polymorphisms have a significant impact on acute rejection in kidney transplantationMaike Janssen, Friedrich Thaiss, Björn Nashan, et al.American Journal of Nephrology|April 4, 2013
Effects of everolimus on oxidative stress in kidney model of ischemia/reperfusion injuryAleksandra Kezic, Friedrich Thaiss, Jan U Becker, et al.Pageof 80