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Nissi Varghese

Showing results (1-10 of 8) with videos related to

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Developmental Neuroscience|April 1, 2021
Flexible Stoichiometry: Implications for KCNQ2- and KCNQ3-Associated Neurodevelopmental DisordersKristen Springer, Nissi Varghese, Anastasios V Tzingounis
Journal of Neurophysiology|March 17, 2021
KCNQ3 is the principal target of retigabine in CA1 and subicular excitatory neuronsNissi Varghese, Anna Lauritano, Maurizio Taglialatela, et al.
Eneuro|April 17, 2021
Loss of KCNQ2 or KCNQ3 Leads to Multifocal Time-Varying Activity in the Neonatal Forebrain <i>Ex Vivo</i>Bowen Hou, Nissi Varghese, Heun Soh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 22, 2023
KCNQ2/3 Gain-of-Function Variants and Cell Excitability: Differential Effects in CA1 versus L2/3 Pyramidal NeuronsNissi Varghese, Bruno Moscoso, Ana Chavez, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 19, 2025
Mislocalization of KCNQ2 Channels as a Pathogenic Mechanism in KCNQ2 Developmental and Epileptic EncephalopathyKristen Springer, Heun Soh, Raquel Paz Zavala, et al.
Neurobiology of Pain (Cambridge, Mass.)|April 11, 2025
Humanized Na<sub>V</sub>1.8 rats overcome cross-species potency shifts in developing novel Na<sub>V</sub>1.8 inhibitorsDillon S McDevitt, Joshua D Vardigan, Xiaoping Zhou, et al.
Elife|January 6, 2025
Plural molecular and cellular mechanisms of pore domain <i>KCNQ2</i> encephalopathyTimothy J Abreo, Emma C Thompson, Anuraag Madabushi, et al.
Biorxiv : the Preprint Server for Biology|January 23, 2024
Plural molecular and cellular mechanisms of pore domain <i>KCNQ2</i> encephalopathyTimothy J Abreo, Emma C Thompson, Anuraag Madabushi, et al.
Pageof 1

Showing results (1-10 of 8) with videos related to

Sort By:
Pageof 1
Developmental Neuroscience|April 1, 2021
Flexible Stoichiometry: Implications for KCNQ2- and KCNQ3-Associated Neurodevelopmental DisordersKristen Springer, Nissi Varghese, Anastasios V Tzingounis
Journal of Neurophysiology|March 17, 2021
KCNQ3 is the principal target of retigabine in CA1 and subicular excitatory neuronsNissi Varghese, Anna Lauritano, Maurizio Taglialatela, et al.
Eneuro|April 17, 2021
Loss of KCNQ2 or KCNQ3 Leads to Multifocal Time-Varying Activity in the Neonatal Forebrain <i>Ex Vivo</i>Bowen Hou, Nissi Varghese, Heun Soh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 22, 2023
KCNQ2/3 Gain-of-Function Variants and Cell Excitability: Differential Effects in CA1 versus L2/3 Pyramidal NeuronsNissi Varghese, Bruno Moscoso, Ana Chavez, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 19, 2025
Mislocalization of KCNQ2 Channels as a Pathogenic Mechanism in KCNQ2 Developmental and Epileptic EncephalopathyKristen Springer, Heun Soh, Raquel Paz Zavala, et al.
Neurobiology of Pain (Cambridge, Mass.)|April 11, 2025
Humanized Na<sub>V</sub>1.8 rats overcome cross-species potency shifts in developing novel Na<sub>V</sub>1.8 inhibitorsDillon S McDevitt, Joshua D Vardigan, Xiaoping Zhou, et al.
Elife|January 6, 2025
Plural molecular and cellular mechanisms of pore domain <i>KCNQ2</i> encephalopathyTimothy J Abreo, Emma C Thompson, Anuraag Madabushi, et al.
Biorxiv : the Preprint Server for Biology|January 23, 2024
Plural molecular and cellular mechanisms of pore domain <i>KCNQ2</i> encephalopathyTimothy J Abreo, Emma C Thompson, Anuraag Madabushi, et al.
Pageof 1