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Niza Frenkel

Showing results (1-10 of 11) with videos related to

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Current Gene Therapy|June 22, 2006
The history of the HSV amplicon: from naturally occurring defective genomes to engineered amplicon vectorsNiza Frenkel
Proceedings of the National Academy of Sciences of the United States of America|October 28, 2009
Cloning human herpes virus 6A genome into bacterial artificial chromosomes and study of DNA replication intermediatesRonen Borenstein, Niza Frenkel
Current Gene Therapy|June 22, 2006
Characterization of the lymphotropic amplicons-6 and tamplicon-7 vectors derived from HHV-6 and HHV-7Niza Frenkel, Ronen Borenstein
Journal of Virology|October 5, 2007
Human herpesvirus 6A (HHV-6A) and HHV-6B alter E2F1/Rb pathways and E2F1 localization and cause cell cycle arrest in infected T cellsGuy Mlechkovich, Niza Frenkel
Journal of Virology|February 17, 2017
Human Herpesvirus 6A Exhibits Restrictive Propagation with Limited Activation of the Protein Kinase R-eIF2α Stress PathwayEyal Sharon, Niza Frenkel
Journal of Virology|January 8, 2010
The DR1 and DR6 first exons of human herpesvirus 6A are not required for virus replication in culture and are deleted in virus stocks that replicate well in T-cell linesRonen Borenstein, Haim Zeigerman, Niza Frenkel
Proceedings of the National Academy of Sciences of the United States of America|December 17, 2013
Human herpesvirus 6 (HHV-6) alters E2F1/Rb pathways and utilizes the E2F1 transcription factor to express viral genesEyal Sharon, Ludmila Volchek, Niza Frenkel
Current Opinion in Virology|December 3, 2014
Roseoloviruses manipulate host cell cycleNiza Frenkel, Eyal Sharon, Haim Zeigerman
Journal of Virology|April 14, 2004
Use of amplicon-6 vectors derived from human herpesvirus 6 for efficient expression of membrane-associated and -secreted proteins in T cellsRonen Borenstein, Oded Singer, Adi Moseri, et al.
Journal of Virology|December 15, 2005
The herpes simplex virus type 1 vhs-UL41 gene secures viral replication by temporarily evading apoptotic cellular response to infection: Vhs-UL41 activity might require interactions with elements of cellular mRNA degradation machineryAri Barzilai, Ifaat Zivony-Elbom, Ronit Sarid, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Current Gene Therapy|June 22, 2006
The history of the HSV amplicon: from naturally occurring defective genomes to engineered amplicon vectorsNiza Frenkel
Proceedings of the National Academy of Sciences of the United States of America|October 28, 2009
Cloning human herpes virus 6A genome into bacterial artificial chromosomes and study of DNA replication intermediatesRonen Borenstein, Niza Frenkel
Current Gene Therapy|June 22, 2006
Characterization of the lymphotropic amplicons-6 and tamplicon-7 vectors derived from HHV-6 and HHV-7Niza Frenkel, Ronen Borenstein
Journal of Virology|October 5, 2007
Human herpesvirus 6A (HHV-6A) and HHV-6B alter E2F1/Rb pathways and E2F1 localization and cause cell cycle arrest in infected T cellsGuy Mlechkovich, Niza Frenkel
Journal of Virology|February 17, 2017
Human Herpesvirus 6A Exhibits Restrictive Propagation with Limited Activation of the Protein Kinase R-eIF2α Stress PathwayEyal Sharon, Niza Frenkel
Journal of Virology|January 8, 2010
The DR1 and DR6 first exons of human herpesvirus 6A are not required for virus replication in culture and are deleted in virus stocks that replicate well in T-cell linesRonen Borenstein, Haim Zeigerman, Niza Frenkel
Proceedings of the National Academy of Sciences of the United States of America|December 17, 2013
Human herpesvirus 6 (HHV-6) alters E2F1/Rb pathways and utilizes the E2F1 transcription factor to express viral genesEyal Sharon, Ludmila Volchek, Niza Frenkel
Current Opinion in Virology|December 3, 2014
Roseoloviruses manipulate host cell cycleNiza Frenkel, Eyal Sharon, Haim Zeigerman
Journal of Virology|April 14, 2004
Use of amplicon-6 vectors derived from human herpesvirus 6 for efficient expression of membrane-associated and -secreted proteins in T cellsRonen Borenstein, Oded Singer, Adi Moseri, et al.
Journal of Virology|December 15, 2005
The herpes simplex virus type 1 vhs-UL41 gene secures viral replication by temporarily evading apoptotic cellular response to infection: Vhs-UL41 activity might require interactions with elements of cellular mRNA degradation machineryAri Barzilai, Ifaat Zivony-Elbom, Ronit Sarid, et al.
Pageof 2