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Heart and Vessels|February 22, 2018
Correction to: High-intensity aerobic interval training can lead to improvement in skeletal muscle power among in-hospital patients with advanced heart failureMasanobu Taya, Eisuke Amiya, Masaru Hatano, et al.Orphanet Journal of Rare Diseases|June 14, 2017
International physician survey on management of FOP: a modified Delphi studyMaja Di Rocco, Genevieve Baujat, Marta Bertamino, et al.European Journal of Human Genetics : EJHG|July 24, 2019
Comprehensive clinical and molecular studies in split-hand/foot malformation: identification of two plausible candidate genes (LRP6 and UBA2)Kaori Yamoto, Hirotomo Saitsu, Gen Nishimura, et al.Geriatrics & Gerontology International|February 19, 2025
Reconstruction of a resilient and secure community and medical care system in the coronavirus era - English translation of the Japanese opinion released from the Science Council of JapanKatsuya Iijima, Masahiro Akishita, Tamao Endo, et al.Biochemical and Biophysical Research Communications|October 28, 2008
A unique mutation of ALK2, G356D, found in a patient with fibrodysplasia ossificans progressiva is a moderately activated BMP type I receptorToru Fukuda, Kazuhiro Kanomata, Junya Nojima, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|December 30, 2022
Reduction of New Heterotopic Ossification (HO) in the Open-Label, Phase 3 MOVE Trial of Palovarotene for Fibrodysplasia Ossificans Progressiva (FOP)Robert J Pignolo, Edward C Hsiao, Mona Al Mukaddam, et al.Clinical Orthopaedics and Related Research|May 8, 2023
Most Fractures Treated Nonoperatively in Individuals With Fibrodysplasia Ossificans Progressiva Heal With a Paucity of Flareups, Heterotopic Ossification, and Loss of MobilityCarter M Lindborg, Mona Al Mukaddam, Genevieve Baujat, et al.Biochemical and Biophysical Research Communications|March 8, 2011
A novel mutation of ALK2, L196P, found in the most benign case of fibrodysplasia ossificans progressiva activates BMP-specific intracellular signaling equivalent to a typical mutation, R206HSatoshi Ohte, Masashi Shin, Hiroki Sasanuma, et al.Bone|February 17, 2020
Self-reported baseline phenotypes from the International Fibrodysplasia Ossificans Progressiva (FOP) Association Global RegistryRobert J Pignolo, Kin Cheung, Sammi Kile, et al.British Journal of Clinical Pharmacology|October 4, 2018
Special considerations for clinical trials in fibrodysplasia ossificans progressiva (FOP)Edward C Hsiao, Maja Di Rocco, Amanda Cali, et al.Pageof 14