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Biochemical and Biophysical Research Communications|July 19, 2011
Bipartite syntaxin 1A interactions mediate CaV2.2 calcium channel regulationJonathan N Davies, Scott E Jarvis, Gerald W Zamponi
Molecular Brain|September 21, 2021
Splice-variant specific effects of a CACNA1H mutation associated with writer's crampIvana A Souza, Maria A Gandini, Gerald W Zamponi
Behavioural Brain Research|March 29, 2011
Depressive-like behaviour of mice lacking cellular prion proteinVinicius M Gadotti, Stephan P Bonfield, Gerald W Zamponi
Molecular Brain|April 30, 2014
Effect of the Brugada syndrome mutation A39V on calmodulin regulation of Cav1.2 channelsBrett A Simms, Ivana Assis Souza, Gerald W Zamponi
Pflugers Archiv : European Journal of Physiology|December 20, 2013
A novel calmodulin site in the Cav1.2 N-terminus regulates calcium-dependent inactivationBrett A Simms, Ivana Assis Souza, Gerald W Zamponi
Molecular Brain|April 19, 2015
Glutamate receptors function as scaffolds for the regulation of β-amyloid and cellular prion protein signaling complexesAlison Hamilton, Gerald W Zamponi, Stephen S G Ferguson
Channels (Austin, Tex.)|August 10, 2013
Modeling interactions between voltage-gated Ca (2+) channels and KCa1.1 channelsJordan Dt Engbers, Gerald W Zamponi, Ray W Turner
Molecular Brain|December 6, 2019
Analgesic effects of optogenetic inhibition of basolateral amygdala inputs into the prefrontal cortex in nerve injured female miceVinicius M Gadotti, Zizhen Zhang, Junting Huang, et al.
Pharmacological Reviews|September 13, 2015
The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialGerald W Zamponi, Joerg Striessnig, Alexandra Koschak, et al.
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