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American Journal of Physiology. Renal Physiology|August 6, 2011
Distinct cardiac and renal effects of ETA receptor antagonist and ACE inhibitor in experimental type 2 diabetesCarla Zoja, Sara Cattaneo, Fabio Fiordaliso, et al.Eclinicalmedicine|January 10, 2022
Nephrotic-range proteinuria in type 2 diabetes: Effects of empagliflozin on kidney disease progression and clinical outcomesPiero Ruggenenti, Bettina J Kraus, Silvio E Inzucchi, et al.Plos One|March 27, 2015
Renal primordia activate kidney regenerative events in a rat model of progressive renal diseaseBarbara Imberti, Daniela Corna, Paola Rizzo, et al.Molecular Immunology|July 23, 2023
Liver factor B silencing to cure C3 glomerulopathy: Evidence from a mouse model of complement dysregulationCristina Zanchi, Monica Locatelli, Daniela Corna, et al.Scientific Reports|March 7, 2015
Renal progenitors derived from human iPSCs engraft and restore function in a mouse model of acute kidney injuryBarbara Imberti, Susanna Tomasoni, Osele Ciampi, et al.Nephron|July 21, 2026
Sustained terminal complement inhibition on the endothelium with ravulizumab in patients with atypical Haemolytic Uremic SyndromeSara Gastoldi, Elena Bresin, Andrea Pasini, et al.Transplantation|June 28, 2011
Both darbepoetin alfa and carbamylated erythropoietin prevent kidney graft dysfunction due to ischemia/reperfusion in ratsPaola Cassis, Nadia Azzollini, Samantha Solini, et al.American Journal of Physiology. Renal Physiology|August 14, 2009
Unlike each drug alone, lisinopril if combined with avosentan promotes regression of renal lesions in experimental diabetesElena Gagliardini, Daniela Corna, Carla Zoja, et al.Journal of the American Society of Nephrology : JASN|September 30, 2003
Protein overload induces fractalkine upregulation in proximal tubular cells through nuclear factor kappaB- and p38 mitogen-activated protein kinase-dependent pathwaysRoberta Donadelli, Cristina Zanchi, Marina Morigi, et al.Thrombosis and Haemostasis|February 24, 2006
Relation between ADAMTS13 activity and ADAMTS13 antigen levels in healthy donors and patients with thrombotic microangiopathies (TMA)Manfred Rieger, Silvia Ferrari, Johanna A Kremer Hovinga, et al.Pageof 78