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International Journal of Cancer|February 20, 2013
Dichloroacetate improves immune dysfunction caused by tumor-secreted lactic acid and increases antitumor immunoreactivityToshimitsu Ohashi, Takashi Akazawa, Mitsuhiro Aoki, et al.International Journal of Molecular Sciences|February 13, 2025
Dynamics of Immune Cell Infiltration and Fibroblast-Derived IL-33/ST2 Axis Induction in a Mouse Model of Post-Surgical LymphedemaKazuhisa Uemura, Kei-Ichi Katayama, Toshihiko Nishioka, et al.International Immunology|December 7, 2010
IL-23-dependent and -independent enhancement pathways of IL-17A production by lactic acidMasahiko Yabu, Hiroaki Shime, Hiromitsu Hara, et al.Biochimica Et Biophysica Acta|March 1, 2016
Collectin CL-P1 utilizes C-reactive protein for complement activationNitai Roy, Katsuki Ohtani, Yasuyuki Matsuda, et al.Immunological Medicine|March 30, 2021
Anti-complement factor H (CFH) antibodies and a novel CFH gene mutation in an atypical hemolytic uremic syndrome patient with complement activation of the classical pathwaySonoko Minato, Hiroyuki Iijima, Hiro Nakao, et al.Brain & Development|April 12, 2026
Clinical validation of CD16b as a standardized biomarker for inherited GPI deficienciesJunpei Tanigawa, Kae Imanishi, Saori Umeshita, et al.International Journal of Cancer|May 3, 2014
Development of a dendritic cell-targeting lipopeptide as an immunoadjuvant that inhibits tumor growth without inducing local inflammationTakashi Akazawa, Toshimitsu Ohashi, Hiroko Nakajima, et al.Molecular Biology of the Cell|June 13, 2003
Human PIG-U and yeast Cdc91p are the fifth subunit of GPI transamidase that attaches GPI-anchors to proteinsYeongjin Hong, Kazuhito Ohishi, Ji Young Kang, et al.Biochimica Et Biophysica Acta. General Subjects|November 20, 2016
Three pentraxins C-reactive protein, serum amyloid p component and pentraxin 3 mediate complement activation using Collectin CL-P1Nitai Roy, Katsuki Ohtani, Yoshihiko Hidaka, et al.Neuropathology and Applied Neurobiology|June 26, 2026
Neuropathological and Molecular Features Associated With a Heterozygous DNAJC7 Mutation in Amyotrophic Lateral SclerosisYoshiaki Nakayama, Kodai Kume, Takashi Baba, et al.Pageof 8