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Molecular Therapy. Nucleic Acids|January 24, 2025
In vivo efficacy and safety of systemically administered serinol nucleic acid-modified antisense oligonucleotides in mouse kidneyToshiki Tsuboi, Keita Hattori, Takuji Ishimoto, et al.
CEN Case Reports|June 29, 2026
Postpartum TMA requiring dialysis with discordant complement tests: a case reportHikari Fujimura, Shun Minatoguchi, Riku Takeuchi, et al.
Clinical and Experimental Nephrology|May 26, 2018
Investigation on the benefits of mycophenolate mofetil and therapeutic drug monitoring in the treatment of Japanese patients with lupus nephritisTakayuki Katsuno, Takenori Ozaki, Takaya Ozeki, et al.
The Journal of Clinical Investigation|May 20, 2009
The growth factor midkine regulates the renin-angiotensin system in miceAkinori Hobo, Yukio Yuzawa, Tomoki Kosugi, et al.
Kidney International Reports|September 22, 2025
Dysregulated Anaerobic Glycolysis in Podocytes is Relevant to the Progression of Focal Segmental GlomerulosclerosisMasahiro Sugimura, Kayaho Maeda, Katsuaki Shibata, et al.
Arthritis & Rheumatology (Hoboken, N.J.)|April 21, 2015
CD147/basigin limits lupus nephritis and Th17 cell differentiation in mice by inhibiting the interleukin-6/STAT-3 pathwayKayaho Maeda, Tomoki Kosugi, Waichi Sato, et al.
The American Journal of Pathology|February 11, 2017
Growth Factor Midkine Promotes T-Cell Activation through Nuclear Factor of Activated T Cells Signaling and Th1 Cell Differentiation in Lupus NephritisTomohiro Masuda, Kayaho Maeda, Waichi Sato, et al.
Molecular Therapy. Nucleic Acids|March 14, 2025
Erratum: In vivo efficacy and safety of systemically administered serinol nucleic acid-modified antisense oligonucleotides in mouse kidneyToshiki Tsuboi, Keita Hattori, Takuji Ishimoto, et al.
The American Journal of Pathology|December 4, 2012
Deficiency of growth factor midkine exacerbates necrotizing glomerular injuries in progressive glomerulonephritisHiroshi Kojima, Tomoki Kosugi, Waichi Sato, et al.
Internal Medicine (Tokyo, Japan)|February 9, 2025
Unexpected Hypotension in a Female Patient with Fabry Disease: Switching from Agalsidase α to β after Long-term ERTTakuya Sugiura, Reiko Muto, Tatsuaki Amano, et al.
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