Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Nyce

Showing results (1-10 of 148) with videos related to

Pageof 15
Sort By:
Cancer Research|November 1, 1989
Drug-induced DNA hypermethylation and drug resistance in human tumorsJ Nyce
Endocrine-Related Cancer|December 2, 2020
Alert to US physicians: DHEA, widely used as an OTC androgen supplement, may exacerbate COVID-19Jonathan Nyce
Somatic Cell and Molecular Genetics|November 1, 1991
Gene silencing in mammalian cells by direct incorporation of electroporated 5-methyl-2'-deoxycytidine 5'-triphosphateJ Nyce
Current Opinion in Allergy and Clinical Immunology|January 31, 2004
Respirable antisense oligonucleotides: a new, third drug class targeting respiratory diseaseJonathan Nyce
Cancer Drug Resistance (Alhambra, Calif.)|May 18, 2022
Components of the human-specific, p53-mediated "kill switch" tumor suppression mechanism are usurped by human tumors, creating the possibility of therapeutic exploitationJonathan Nyce
Endocrine-Related Cancer|December 10, 2019
A lex naturalis delineates components of a human-specific, adrenal androgen-dependent, p53-mediated 'kill switch' tumor suppression mechanismJonathan Wesley Nyce
Trends in Pharmacological Sciences|April 2, 1999
Insight into adenosine receptor function using antisense and gene-knockout approachesJ W Nyce
Endocrine-Related Cancer|November 8, 2018
Species-specific mechanisms of tumor suppression are fundamental drivers of vertebrate speciation: critical implications for the 'war on cancer'Jonathan W Nyce
Angewandte Chemie (International Ed. in English)|January 29, 2000
Formal Three-Electron Reduction by an f-Element Complex: Formation ofEvans, Nyce, Ziller
Endocrine-Related Cancer|June 27, 2018
Detection of a novel, primate-specific 'kill switch' tumor suppression mechanism that may fundamentally control cancer risk in humans: an unexpected twist in the basic biology of TP53Jonathan W Nyce
Pageof 15

Showing results (1-10 of 148) with videos related to

Sort By:
Pageof 15
Cancer Research|November 1, 1989
Drug-induced DNA hypermethylation and drug resistance in human tumorsJ Nyce
Endocrine-Related Cancer|December 2, 2020
Alert to US physicians: DHEA, widely used as an OTC androgen supplement, may exacerbate COVID-19Jonathan Nyce
Somatic Cell and Molecular Genetics|November 1, 1991
Gene silencing in mammalian cells by direct incorporation of electroporated 5-methyl-2'-deoxycytidine 5'-triphosphateJ Nyce
Current Opinion in Allergy and Clinical Immunology|January 31, 2004
Respirable antisense oligonucleotides: a new, third drug class targeting respiratory diseaseJonathan Nyce
Cancer Drug Resistance (Alhambra, Calif.)|May 18, 2022
Components of the human-specific, p53-mediated "kill switch" tumor suppression mechanism are usurped by human tumors, creating the possibility of therapeutic exploitationJonathan Nyce
Endocrine-Related Cancer|December 10, 2019
A lex naturalis delineates components of a human-specific, adrenal androgen-dependent, p53-mediated 'kill switch' tumor suppression mechanismJonathan Wesley Nyce
Trends in Pharmacological Sciences|April 2, 1999
Insight into adenosine receptor function using antisense and gene-knockout approachesJ W Nyce
Endocrine-Related Cancer|November 8, 2018
Species-specific mechanisms of tumor suppression are fundamental drivers of vertebrate speciation: critical implications for the 'war on cancer'Jonathan W Nyce
Angewandte Chemie (International Ed. in English)|January 29, 2000
Formal Three-Electron Reduction by an f-Element Complex: Formation ofEvans, Nyce, Ziller
Endocrine-Related Cancer|June 27, 2018
Detection of a novel, primate-specific 'kill switch' tumor suppression mechanism that may fundamentally control cancer risk in humans: an unexpected twist in the basic biology of TP53Jonathan W Nyce
Pageof 15