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Cell Death & Disease|July 18, 2014
Ischemic tolerance modulates TRAIL expression and its receptors and generates a neuroprotected phenotypeG Cantarella, G Pignataro, G Di Benedetto, et al.European Journal of Heart Failure|December 5, 2024
Outpatient worsening of heart failure and mortality in transthyretin amyloid cardiomyopathyLawrence Zeldin, Joanna B S Eichler, Sergio L Teruya, et al.CNS Neuroscience & Therapeutics|September 18, 2023
miR135a administration ameliorates brain ischemic damage by preventing TRPM7 activation during brain ischemiaP Cepparulo, P Brancaccio, R Sirabella, et al.Annals of the New York Academy of Sciences|April 21, 2007
ncx1, ncx2, and ncx3 gene product expression and function in neuronal anoxia and brain ischemiaL Annunziato, G Pignataro, F Boscia, et al.ACI Open|June 9, 2025
Stakeholder Perspectives on the Meaningful Integration of Clinical Informatics Interventions Using Patient-Reported Outcomes in HealthcareDavid Russell, Yashika Sharma, Andrew P Ambrosy, et al.JACC. Cardiovascular Imaging|May 29, 2025
Early Detection of Transthyretin Cardiac Amyloidosis Using 124I-Evuzamitide Positron Emission Tomography/Computed TomographyDia A Smiley, Andrew J Einstein, Kevin J O'Gorman, et al.Clinical Transplantation|July 6, 2020
United network for organ sharing outcomes after heart transplantation for al compared to ATTR cardiac amyloidosisJan M Griffin, Leonard Chiu, Kelly M Axsom, et al.Pageof 4