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Cerebral Cortex (New York, N.Y. : 1991)|June 16, 2011
Involvement of cannabinoid CB1 receptor in associative learning and in hippocampal CA3-CA1 synaptic plasticityNoelia Madroñal, Agnès Gruart, Olga Valverde, et al.
Plos One|November 14, 2015
Reduced Contextual Discrimination following Alcohol Consumption or MDMA Administration in MiceEmily M Johansson, María S García-Gutiérrez, María Moscoso-Castro, et al.
Life Sciences|February 2, 2019
Binge ethanol and MDMA combination exacerbates HSP27 and Trx-1 (biomarkers of toxic cardiac effects) expression in right ventricleJavier Navarro-Zaragoza, Clara Ros-Simó, María-Victoria Milanés, et al.
The European Journal of Neuroscience|September 29, 2004
Adenosine A2A receptors are involved in physical dependence and place conditioning induced by THCGuadalupe Soria, Anna Castañé, Fernando Berrendero, et al.
Journal of Neurochemistry|October 20, 2007
CB1 knockout mice display impaired functionality of 5-HT1A and 5-HT2A/C receptorsSusana Mato, Ester Aso, Elena Castro, et al.
Neuropharmacology|December 3, 2020
Early-life stress exacerbates the effects of WIN55,212-2 and modulates the cannabinoid receptor type 1 expressionAna Martín-Sánchez, Alba García-Baos, Adriana Castro-Zavala, et al.
Progress in Neuro-Psychopharmacology & Biological Psychiatry|January 26, 2021
Cocaine-seeking behaviour is differentially expressed in male and female mice exposed to maternal separation and is associated with alterations in AMPA receptors subunits in the medial prefrontal cortexAdriana Castro-Zavala, Ana Martín-Sánchez, Larisa Montalvo-Martínez, et al.
Neuropharmacology|September 20, 2005
Development and expression of neuropathic pain in CB1 knockout miceAnna Castañé, Evelyne Célérier, Miquel Martín, et al.
Plos One|October 29, 2015
Binge Ethanol and MDMA Combination Exacerbates Toxic Cardiac Effects by Inducing Cellular StressJavier Navarro-Zaragoza, Clara Ros-Simó, María-Victoria Milanés, et al.
Scientific Reports|October 25, 2023
Cocaine-induced loss of LTD and social impairments are restored by fatty acid amide hydrolase inhibitionLaia Alegre-Zurano, Alba Caceres-Rodriguez, Paula Berbegal-Sáez, et al.
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