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Disease Models & Mechanisms|January 7, 2021
A muscle growth-promoting treatment based on the attenuation of activin/myostatin signalling results in long-term testicular abnormalitiesDanielle Vaughan, Robert Mitchell, Oliver Kretz, et al.The Journal of Cell Biology|December 9, 2015
The polarity protein Inturned links NPHP4 to Daam1 to control the subapical actin network in multiciliated cellsTakayuki Yasunaga, Sylvia Hoff, Christoph Schell, et al.Nature Cell Biology|November 8, 2016
Direct reprogramming of fibroblasts into renal tubular epithelial cells by defined transcription factorsMichael M Kaminski, Jelena Tosic, Catena Kresbach, et al.European Journal of Translational Myology|June 6, 2020
Inhibition of Activin/Myostatin signalling induces skeletal muscle hypertrophy but impairs mouse testicular developmentDanielle Vaughan, Olli Ritvos, Robert Mitchell, et al.Journal of Molecular Cell Biology|June 18, 2015
The GYF domain protein CD2BP2 is critical for embryogenesis and podocyte functionGesa I Albert, Christoph Schell, Karin M Kirschner, et al.Acta Physiologica (Oxford, England)|May 27, 2020
Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndromeMengyun Xiao, Bernhard N Bohnert, Hande Aypek, et al.Kidney International|October 3, 2022
Podocyte expression of human phospholipase A2 receptor 1 causes immune-mediated membranous nephropathy in miceNicola M Tomas, Silke Dehde, Catherine Meyer-Schwesinger, et al.European Journal of Translational Myology|August 13, 2020
Diminution in sperm quantity and quality in mouse models of Duchenne Muscular Dystrophy induced by a myostatin-based muscle growth-promoting interventionDanielle Vaughan, Oliver Kretz, Ali Alqallaf, et al.Cell|October 22, 2016
MOF Acetyl Transferase Regulates Transcription and Respiration in MitochondriaAindrila Chatterjee, Janine Seyfferth, Jacopo Lucci, et al.JCI Insight|February 26, 2026
Functional characterization of podocyte-expressed THSD7A in experimental membranous nephropathyMing Huang, Moritz Lassé, Silke Dehde, et al.Pageof 11