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BMC Molecular Biology|September 21, 2007
XRCC1 and PCNA are loading platforms with distinct kinetic properties and different capacities to respond to multiple DNA lesionsOliver Mortusewicz, Heinrich LeonhardtJournal of Cellular Biochemistry|April 4, 2008
Spatiotemporal dynamics of regulatory protein recruitment at DNA damage sitesOliver Mortusewicz, Heinrich Leonhardt, M Cristina CardosoBiomolecules|October 29, 2020
OGG1 Inhibitor TH5487 Alters OGG1 Chromatin Dynamics and Prevents IncisionsBishoy M F Hanna, Thomas Helleday, Oliver MortusewiczMolecular Cell|May 16, 2025
Uracil-induced replication stress drives mutations, genome instability, anti-cancer treatment efficacy, and resistanceOliver Mortusewicz, James Haslam, Helge Gad, et al.Nucleic Acids Research|November 6, 2007
Feedback-regulated poly(ADP-ribosyl)ation by PARP-1 is required for rapid response to DNA damage in living cellsOliver Mortusewicz, Jean-Christophe Amé, Valérie Schreiber, et al.Nucleic Acids Research|July 21, 2006
Differential recruitment of DNA Ligase I and III to DNA repair sitesOliver Mortusewicz, Ulrich Rothbauer, M Cristina Cardoso, et al.International Journal of Molecular Sciences|April 30, 2021
NEIL1 and NEIL2 Are Recruited as Potential Backup for OGG1 upon OGG1 Depletion or Inhibition by TH5487Bishoy M F Hanna, Maurice Michel, Thomas Helleday, et al.Nucleic Acids Research|March 15, 2011
PARG is recruited to DNA damage sites through poly(ADP-ribose)- and PCNA-dependent mechanismsOliver Mortusewicz, Elise Fouquerel, Jean-Christophe Amé, et al.Proceedings of the National Academy of Sciences of the United States of America|June 16, 2005
Recruitment of DNA methyltransferase I to DNA repair sitesOliver Mortusewicz, Lothar Schermelleh, Joachim Walter, et al.The Journal of Cell Biology|December 3, 2008
Recruitment of RNA polymerase II cofactor PC4 to DNA damage sitesOliver Mortusewicz, Wera Roth, Na Li, et al.Pageof 4