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Acta Neuropathologica|March 8, 2006
Axonopathy in an APP/PS1 transgenic mouse model of Alzheimer's diseaseOliver Wirths, Joachim Weis, Jacek Szczygielski, et al.
Journal of Alzheimer'S Disease : JAD|March 14, 2013
Oligomeric pyroglutamate amyloid-β is present in microglia and a subfraction of vessels in patients with Alzheimer's disease: implications for immunotherapyOliver Wirths, Antje Hillmann, Laurent Pradier, et al.
International Journal of Molecular Sciences|February 11, 2023
A Combination of Caffeine Supplementation and Enriched Environment in an Alzheimer's Disease Mouse ModelMartina Stazi, Silvia Zampar, Hans-Wolfgang Klafki, et al.
Neurobiology of Aging|July 13, 2010
Motor deficits, neuron loss, and reduced anxiety coinciding with axonal degeneration and intraneuronal Aβ aggregation in the 5XFAD mouse model of Alzheimer's diseaseSadim Jawhar, Anna Trawicka, Carolin Jenneckens, et al.
Neuro-Degenerative Diseases|March 7, 2008
Intraneuronal beta-amyloid is a major risk factor--novel evidence from the APP/PS1KI mouse modelThomas A Bayer, Henning Breyhan, Kailai Duan, et al.
CNS Neuroscience & Therapeutics|February 5, 2016
The Cannabinoid CB1/CB2 Agonist WIN55212.2 Promotes Oligodendrocyte Differentiation In Vitro and Neuroprotection During the Cuprizone-Induced Central Nervous System DemyelinationJordi Tomas-Roig, Oliver Wirths, Gabriela Salinas-Riester, et al.
Brain Research|April 13, 2007
Altered cholesterol metabolism in APP695-transfected neuroblastoma cellsOliver Wirths, Karin M Thelen, Dieter Lütjohann, et al.
Neurobiology of Aging|September 12, 2006
Age-dependent axonal degeneration in an Alzheimer mouse modelOliver Wirths, Joachim Weis, Rakez Kayed, et al.
European Archives of Psychiatry and Clinical Neuroscience|June 8, 2022
Combined long-term enriched environment and caffeine supplementation improve memory function in C57Bl6 miceMartina Stazi, Silvia Zampar, Madeleine Nadolny, et al.
Biochimica Et Biophysica Acta. Molecular Cell Research|August 14, 2021
The anti-parallel dimer binding interface in STAT3 transcription factor is required for the inactivation of cytokine-mediated signal transductionPriyanka Rajeev Menon, Asmma Doudin, Anke Gregus, et al.
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