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Molecular Neurobiology|May 15, 2026
Amyloid Plaques Ameliorate Memory Deficits and Hippocampal Neuron Loss in an Aβ4-42-Driven Alzheimer's Disease Mouse ModelSilvia Zampar, Merle Fricke, Florian Kremser, et al.
Brain, Behavior, and Immunity|December 27, 2024
Cognitive decline and neuroinflammation in a mouse model of obesity: An accelerating role of ageingMansi Rajput, Ihtzaz Ahmed Malik, Aditi Methi, et al.
Life (Basel, Switzerland)|April 28, 2023
Brain Region-Specific Differences in Amyloid-β Plaque Composition in 5XFAD MiceAngelika Sabine Bader, Marius-Uwe Gnädig, Merle Fricke, et al.
The American Journal of Pathology|February 7, 2012
Amyloid precursor protein is a biomarker for transformed human pluripotent stem cellsVivek Venkataramani, Knut Thiele, Carl Ludwig Behnes, et al.
The World Journal of Biological Psychiatry : the Official Journal of the World Federation of Societies of Biological Psychiatry|March 12, 2010
Gene expression of neuregulin-1 isoforms in different brain regions of elderly schizophrenia patientsEleni Parlapani, Andrea Schmitt, Oliver Wirths, et al.
The Journal of Biological Chemistry|January 24, 2012
Pyroglutamate amyloid β (Aβ) aggravates behavioral deficits in transgenic amyloid mouse model for Alzheimer diseaseJessica L Wittnam, Erik Portelius, Henrik Zetterberg, et al.
Journal of Alzheimer'S Disease : JAD|September 4, 2014
I716F AβPP mutation associates with the deposition of oligomeric pyroglutamate amyloid-β and α-synucleinopathy with Lewy bodiesEvelyn Sieczkowski, Ivan Milenkovic, Vivek Venkataramani, et al.
Frontiers in Aging Neuroscience|March 24, 2018
Synergistic Effect on Neurodegeneration by N-Truncated Aβ<sub>4-42</sub> and Pyroglutamate Aβ<sub>3-42</sub> in a Mouse Model of Alzheimer's DiseaseJose S Lopez-Noguerola, Nicolai M E Giessen, Maximilian Ueberück, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|October 31, 2008
Effect of copper intake on CSF parameters in patients with mild Alzheimer's disease: a pilot phase 2 clinical trialHolger Kessler, Frank-Gerald Pajonk, Daniela Bach, et al.
Acta Neuropathologica|May 21, 2013
N-truncated amyloid β (Aβ) 4-42 forms stable aggregates and induces acute and long-lasting behavioral deficitsYvonne Bouter, Katharina Dietrich, Jessica L Wittnam, et al.
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