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Current Opinion in Clinical Nutrition and Metabolic Care|April 12, 2006
Gene therapy with anabolic growth factors to prevent muscle atrophyOlivier Schakman, Jean-Paul ThissenMolecular and Cellular Neurosciences|May 15, 2013
The Onecut transcription factor HNF-6 contributes to proper reorganization of Purkinje cells during postnatal cerebellum developmentEmilie Audouard, Olivier Schakman, Audrey Ginion, et al.FEBS Letters|October 21, 2009
Essential role of TRPV2 ion channel in the sensitivity of dystrophic muscle to eccentric contractionsNadège Zanou, Yuko Iwata, Olivier Schakman, et al.Frontiers in Molecular Neuroscience|February 3, 2023
Inhibitory synapse dysfunction and epileptic susceptibility associated with KIF2A deletion in cortical interneuronsNuria Ruiz-Reig, Dario García-Sánchez, Olivier Schakman, et al.Magnetic Resonance in Medicine|March 24, 2012
Hexafluorobenzene in comparison with perfluoro-15-crown-5-ether for repeated monitoring of oxygenation using 19F MRI in a mouse modelLionel Mignion, Julie Magat, Olivier Schakman, et al.American Journal of Physiology. Endocrinology and Metabolism|May 14, 2009
Follistatin induces muscle hypertrophy through satellite cell proliferation and inhibition of both myostatin and activinHélène Gilson, Olivier Schakman, Stéphanie Kalista, et al.The Journal of Biological Chemistry|March 9, 2012
Trpc1 ion channel modulates phosphatidylinositol 3-kinase/Akt pathway during myoblast differentiation and muscle regenerationNadège Zanou, Olivier Schakman, Pierre Louis, et al.The Journal of Gene Medicine|October 6, 2012
Multiparametric functional nuclear magnetic resonance imaging shows alterations associated with plasmid electrotransfer in mouse skeletal muscleCeline Baligand, Gregory Jouvion, Olivier Schakman, et al.Iscience|August 8, 2025
Conditional deletion of TRPC1 channel modulates synaptic plasticity, long term depression, and memory extinction in Fragile X syndrome miceFarah Issa, Xavier Yerna, Thibaud Parpaite, et al.Metabolic Brain Disease|July 9, 2015
Early ALS-type gait abnormalities in AMP-dependent protein kinase-deficient mice suggest a role for this metabolic sensor in early stages of the diseaseMaxime Vergouts, Claudia Marinangeli, Caroline Ingelbrecht, et al.Pageof 5