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Plos One|April 19, 2012
Impaired adaptive response to mechanical overloading in dystrophic skeletal musclePierre Joanne, Christophe Hourdé, Julien Ochala, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|June 27, 2014
Myostatin is a key mediator between energy metabolism and endurance capacity of skeletal muscleEtienne Mouisel, Karima Relizani, Laurence Mille-Hamard, et al.
Journal of the American College of Cardiology|July 21, 2004
Comparison of human skeletal myoblasts and bone marrow-derived CD133+ progenitors for the repair of infarcted myocardiumOnnik Agbulut, Susanne Vandervelde, Nawwar Al Attar, et al.
European Heart Journal|May 20, 2014
Towards a clinical use of human embryonic stem cell-derived cardiac progenitors: a translational experiencePhilippe Menasché, Valérie Vanneaux, Jean-Roch Fabreguettes, et al.
Plos One|September 15, 2021
Dexamethasone is associated with early deaths in light chain amyloidosis patients with severe cardiac involvementMélanie Bézard, Silvia Oghina, Damien Vitiello, et al.
Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|November 26, 2025
Oxytocin treatment reduces cancer cachexia in a pre-clinical modelAlexandra Sviercovich, Etsuko Watanabe, Estefania S Fernandez, et al.
The EMBO Journal|November 14, 2018
Lipin1 deficiency causes sarcoplasmic reticulum stress and chaperone-responsive myopathyTalha Rashid, Ivan Nemazanyy, Cecilia Paolini, et al.
European Journal of Cell Biology|July 10, 2025
Integrated phenotypic and transcriptomic characterization of desmin-related cardiomyopathy in hiPSC-derived cardiomyocytes and machine learning-based classification of disease featuresVivien Batoumeni, Yeranuhi Hovhannisyan, Bénédicte Gobert, et al.
Journal of the American College of Cardiology|February 2, 2018
Transplantation of Human Embryonic Stem Cell-Derived Cardiovascular Progenitors for Severe Ischemic Left Ventricular DysfunctionPhilippe Menasché, Valérie Vanneaux, Albert Hagège, et al.
Stem Cell Research & Therapy|May 1, 2026
CAR-T cells targeting fibroblast activation protein eliminate pathological fibroblasts and preserve cardiac function in a Duchenne Muscular Dystrophy murine modelCéline Marigny, Gaëlle Revet, Anne Berger, et al.
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