Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Oscar D Bello

Showing results (1-10 of 13) with videos related to

Pageof 2
Sort By:
Langmuir : the ACS Journal of Surfaces and Colloids|March 15, 2016
Using ApoE Nanolipoprotein Particles To Analyze SNARE-Induced Fusion PoresOscar D Bello, Sarah M Auclair, James E Rothman, et al.
Experimental Cell Research|January 18, 2012
RIM, Munc13, and Rab3A interplay in acrosomal exocytosisOscar D Bello, M Natalia Zanetti, Luis S Mayorga, et al.
Elife|August 30, 2017
Circular oligomerization is an intrinsic property of synaptotagminJing Wang, Feng Li, Oscar D Bello, et al.
Elife|March 28, 2017
Dilation of fusion pores by crowding of SNARE proteinsZhenyong Wu, Oscar D Bello, Sathish Thiyagarajan, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2017
Kv1.1 channelopathy abolishes presynaptic spike width modulation by subthreshold somatic depolarizationUmesh Vivekananda, Pavel Novak, Oscar D Bello, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 25, 2018
Synaptotagmin oligomerization is essential for calcium control of regulated exocytosisOscar D Bello, Ouardane Jouannot, Arunima Chaudhuri, et al.
Elife|July 20, 2016
Ring-like oligomers of Synaptotagmins and related C2 domain proteinsMaria N Zanetti, Oscar D Bello, Jing Wang, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 5, 2020
Synaptotagmin 1 oligomers clamp and regulate different modes of neurotransmitter releaseErica Tagliatti, Oscar D Bello, Philipe R F Mendonça, et al.
Human Mutation|November 12, 2017
A loss-of-function homozygous mutation in DDX59 implicates a conserved DEAD-box RNA helicase in nervous system development and functionVincenzo Salpietro, Stephanie Efthymiou, Andreea Manole, et al.
Annals of Neurology|March 3, 2017
Homozygous mutations in VAMP1 cause a presynaptic congenital myasthenic syndromeVincenzo Salpietro, Weichun Lin, Andrea Delle Vedove, et al.
Pageof 2

Showing results (1-10 of 13) with videos related to

Sort By:
Pageof 2
Langmuir : the ACS Journal of Surfaces and Colloids|March 15, 2016
Using ApoE Nanolipoprotein Particles To Analyze SNARE-Induced Fusion PoresOscar D Bello, Sarah M Auclair, James E Rothman, et al.
Experimental Cell Research|January 18, 2012
RIM, Munc13, and Rab3A interplay in acrosomal exocytosisOscar D Bello, M Natalia Zanetti, Luis S Mayorga, et al.
Elife|August 30, 2017
Circular oligomerization is an intrinsic property of synaptotagminJing Wang, Feng Li, Oscar D Bello, et al.
Elife|March 28, 2017
Dilation of fusion pores by crowding of SNARE proteinsZhenyong Wu, Oscar D Bello, Sathish Thiyagarajan, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2017
Kv1.1 channelopathy abolishes presynaptic spike width modulation by subthreshold somatic depolarizationUmesh Vivekananda, Pavel Novak, Oscar D Bello, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 25, 2018
Synaptotagmin oligomerization is essential for calcium control of regulated exocytosisOscar D Bello, Ouardane Jouannot, Arunima Chaudhuri, et al.
Elife|July 20, 2016
Ring-like oligomers of Synaptotagmins and related C2 domain proteinsMaria N Zanetti, Oscar D Bello, Jing Wang, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 5, 2020
Synaptotagmin 1 oligomers clamp and regulate different modes of neurotransmitter releaseErica Tagliatti, Oscar D Bello, Philipe R F Mendonça, et al.
Human Mutation|November 12, 2017
A loss-of-function homozygous mutation in DDX59 implicates a conserved DEAD-box RNA helicase in nervous system development and functionVincenzo Salpietro, Stephanie Efthymiou, Andreea Manole, et al.
Annals of Neurology|March 3, 2017
Homozygous mutations in VAMP1 cause a presynaptic congenital myasthenic syndromeVincenzo Salpietro, Weichun Lin, Andrea Delle Vedove, et al.
Pageof 2