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The American Journal of Physiology|July 17, 1999
Autoimmune gastritis results in disruption of gastric epithelial cell developmentL M Judd, P A Gleeson, B H Toh, et al.
The Journal of Biological Chemistry|December 29, 1998
Expression of three Caenorhabditis elegans N-acetylglucosaminyltransferase I genes during developmentS Chen, S Zhou, M Sarkar, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|December 24, 1982
Oligosaccharide branching of glycoproteins: biosynthetic mechanisms and possible biological functionsH Schachter, S Narasimhan, P Gleeson, et al.
Canadian Journal of Biochemistry and Cell Biology = Revue Canadienne De Biochimie Et Biologie Cellulaire|December 1, 1983
Mucin synthesis. III. UDP-GlcNAc:Gal beta 1-3(GlcNAc beta 1-6)GalNAc-R (GlcNAc to Gal) beta 3-N-acetylglucosaminyltransferase, an enzyme in porcine gastric mucosa involved in the elongation of mucin-type oligosaccharidesI Brockhausen, D Williams, K L Matta, et al.
International Immunology|March 4, 2000
Tolerance and autoimmunity to a gastritogenic peptide in TCR transgenic miceF Alderuccio, V Cataldo, I R van Driel, et al.
Autoimmunity|March 22, 2002
The role of natural killer cells in the induction of autoimmune gastritisN L La Gruta, I R Van Driel, B H Toh, et al.
Journal of Cell Science|May 13, 1999
The Golgi-targeting sequence of the peripheral membrane protein p230L Kjer-Nielsen, C van Vliet, R Erlich, et al.
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