P A Solski - JoVE Visualize

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Journal of Immunology (Baltimore, Md. : 1950)|May 9, 2000

Modulation of HIV-1 replication by a novel RhoA effector activityL Wang, H Zhang, P A Solski, et al.

Biochemical Society Transactions|October 1, 1989

Activation of cellular p21ras by myristoylationJ E Buss, P A Solski, J P Schaeffer, et al.

Science (New York, N.Y.)|March 24, 1989

Activation of the cellular proto-oncogene product p21Ras by addition of a myristylation signalJ E Buss, P A Solski, J P Schaeffer, et al.

Molecular and Cellular Biology|October 1, 1994

Dbl and Vav mediate transformation via mitogen-activated protein kinase pathways that are distinct from those activated by oncogenic RasR Khosravi-Far, M Chrzanowska-Wodnicka, P A Solski, et al.

Oncogene|June 15, 1995

Critical role of Rho in cell transformation by oncogenic RasG C Prendergast, R Khosravi-Far, P A Solski, et al.

Proceedings of the National Academy of Sciences of the United States of America|April 1, 1990

Farnesol modification of Kirsten-ras exon 4B protein is essential for transformationJ H Jackson, C G Cochrane, J R Bourne, et al.

The Journal of Biological Chemistry|January 9, 1999

A non-farnesylated Ha-Ras protein can be palmitoylated and trigger potent differentiation and transformationM A Booden, T L Baker, P A Solski, et al.

Oncogene|August 13, 1999

Cellular functions of TC10, a Rho family GTPase: regulation of morphology, signal transduction and cell growthG A Murphy, P A Solski, S A Jillian, et al.

The Journal of Biological Chemistry|April 5, 1996

Oncogenic Neu/ErbB-2 increases ets, AP-1, and NF-kappaB-dependent gene expression, and inhibiting ets activation blocks Neu-mediated cellular transformationC K Galang, J García-Ramírez, P A Solski, et al.

Molecular and Cellular Biology|July 1, 1996

Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformationR Khosravi-Far, M A White, J K Westwick, et al.

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Showing results (11-20 of 23) with videos related to

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Journal of Immunology (Baltimore, Md. : 1950)|May 9, 2000

Modulation of HIV-1 replication by a novel RhoA effector activityL Wang, H Zhang, P A Solski, et al.

Biochemical Society Transactions|October 1, 1989

Activation of cellular p21ras by myristoylationJ E Buss, P A Solski, J P Schaeffer, et al.

Science (New York, N.Y.)|March 24, 1989

Activation of the cellular proto-oncogene product p21Ras by addition of a myristylation signalJ E Buss, P A Solski, J P Schaeffer, et al.

Molecular and Cellular Biology|October 1, 1994

Dbl and Vav mediate transformation via mitogen-activated protein kinase pathways that are distinct from those activated by oncogenic RasR Khosravi-Far, M Chrzanowska-Wodnicka, P A Solski, et al.

Oncogene|June 15, 1995

Critical role of Rho in cell transformation by oncogenic RasG C Prendergast, R Khosravi-Far, P A Solski, et al.

Proceedings of the National Academy of Sciences of the United States of America|April 1, 1990

Farnesol modification of Kirsten-ras exon 4B protein is essential for transformationJ H Jackson, C G Cochrane, J R Bourne, et al.

The Journal of Biological Chemistry|January 9, 1999

A non-farnesylated Ha-Ras protein can be palmitoylated and trigger potent differentiation and transformationM A Booden, T L Baker, P A Solski, et al.

Oncogene|August 13, 1999

Cellular functions of TC10, a Rho family GTPase: regulation of morphology, signal transduction and cell growthG A Murphy, P A Solski, S A Jillian, et al.

The Journal of Biological Chemistry|April 5, 1996

Oncogenic Neu/ErbB-2 increases ets, AP-1, and NF-kappaB-dependent gene expression, and inhibiting ets activation blocks Neu-mediated cellular transformationC K Galang, J García-Ramírez, P A Solski, et al.

Molecular and Cellular Biology|July 1, 1996

Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformationR Khosravi-Far, M A White, J K Westwick, et al.

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