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P Arese

Showing results (51-60 of 97) with videos related to

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Thrombosis Research|December 15, 1984
Chemical modification of cytoskeletal proteins of human blood platelets by diamideP Spangenberg, R Heller, A Bosia, et al.
Blood|August 1, 1985
Increased red cell calcium, decreased calcium adenosine triphosphatase, and altered membrane proteins during fava bean hemolysis in glucose-6-phosphate dehydrogenase-deficient (Mediterranean variant) individualsF Turrini, A Naitana, L Mannuzzu, et al.
Parasitology|August 21, 2001
Phagocytosis of malarial pigment haemozoin by human monocytes: a confocal microscopy studyE Schwarzer, G Bellomo, G Giribaldi, et al.
European Journal of Clinical Investigation|January 1, 1973
Red cell glycolysis in a case of 3-phosphoglycerate kinase deficiencyP Arese, A Bosia, E Gallo, et al.
FEBS Letters|October 12, 1992
Enhanced IgG- and complement-independent phagocytosis of sulfatide-enriched human erythrocytes by human monocytesM V Serra, F Mannu, A Matera, et al.
Blood Cells|January 1, 1988
Naturally occurring anti-band 3 antibodies and complement in phagocytosis of oxidatively-stressed and in clearance of senescent red cellsH U Lutz, S Fasler, P Stammler, et al.
Blood|June 1, 1993
Characterization of the autologous antibodies that opsonize erythrocytes with clustered integral membrane proteinsF Turrini, F Mannu, P Arese, et al.
The Journal of Biological Chemistry|December 25, 1989
Platelet-activating factor enhances complement-dependent phagocytosis of diamide-treated erythrocytes by human monocytes through activation of protein kinase C and phosphorylation of complement receptor type one (CR1)F Bussolino, E Fischer, F Turrini, et al.
FEBS Letters|June 17, 1996
Increased levels of 4-hydroxynonenal in human monocytes fed with malarial pigment hemozoin. A possible clue for hemozoin toxicityE Schwarzer, O Müller, P Arese, et al.
Toxicologic Pathology|January 1, 1984
Mechanism of action of divicine in a cell-free system and in glucose-6-phosphate dehydrogenase-deficient red cellsM A Baker, A Bosia, G Pescarmona, et al.
Pageof 10

Showing results (51-60 of 97) with videos related to

Sort By:
Pageof 10
Thrombosis Research|December 15, 1984
Chemical modification of cytoskeletal proteins of human blood platelets by diamideP Spangenberg, R Heller, A Bosia, et al.
Blood|August 1, 1985
Increased red cell calcium, decreased calcium adenosine triphosphatase, and altered membrane proteins during fava bean hemolysis in glucose-6-phosphate dehydrogenase-deficient (Mediterranean variant) individualsF Turrini, A Naitana, L Mannuzzu, et al.
Parasitology|August 21, 2001
Phagocytosis of malarial pigment haemozoin by human monocytes: a confocal microscopy studyE Schwarzer, G Bellomo, G Giribaldi, et al.
European Journal of Clinical Investigation|January 1, 1973
Red cell glycolysis in a case of 3-phosphoglycerate kinase deficiencyP Arese, A Bosia, E Gallo, et al.
FEBS Letters|October 12, 1992
Enhanced IgG- and complement-independent phagocytosis of sulfatide-enriched human erythrocytes by human monocytesM V Serra, F Mannu, A Matera, et al.
Blood Cells|January 1, 1988
Naturally occurring anti-band 3 antibodies and complement in phagocytosis of oxidatively-stressed and in clearance of senescent red cellsH U Lutz, S Fasler, P Stammler, et al.
Blood|June 1, 1993
Characterization of the autologous antibodies that opsonize erythrocytes with clustered integral membrane proteinsF Turrini, F Mannu, P Arese, et al.
The Journal of Biological Chemistry|December 25, 1989
Platelet-activating factor enhances complement-dependent phagocytosis of diamide-treated erythrocytes by human monocytes through activation of protein kinase C and phosphorylation of complement receptor type one (CR1)F Bussolino, E Fischer, F Turrini, et al.
FEBS Letters|June 17, 1996
Increased levels of 4-hydroxynonenal in human monocytes fed with malarial pigment hemozoin. A possible clue for hemozoin toxicityE Schwarzer, O Müller, P Arese, et al.
Toxicologic Pathology|January 1, 1984
Mechanism of action of divicine in a cell-free system and in glucose-6-phosphate dehydrogenase-deficient red cellsM A Baker, A Bosia, G Pescarmona, et al.
Pageof 10